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HCV-associated mixed cryoglobulinemia and b-cell non-Hodgkin's lymphoma - pathogenetically related problems

S Yu Milovanova1, L V Lysenko Kozlovskaya1, L Yu Milovanova1

  • 1I.M. Sechenov First Moscow State Medical Univesity, Ministry Health of Russia (Sechenov University), Moscow, Russia.

Terapevticheskii Arkhiv
|February 1, 2019
PubMed

Insights

Hepatitis C virus (HCV) significantly increases the risk of developing non-Hodgkin's lymphoma (NHL). Antiviral therapy shows effectiveness in treating HCV-associated NHL, offering a crucial treatment avenue.

Area of Science:

  • Hepatology
  • Oncology
  • Immunology

Background:

  • Hepatitis C virus (HCV) infection is a global health concern with significant hepatic and extrahepatic manifestations.
  • Mixed cryoglobulinemia (CG), an extrahepatic manifestation of HCV, is recognized as a B-cell lymphoproliferative disorder.
  • Epidemiological studies and treatment outcomes suggest a link between HCV and lymphoproliferative diseases like non-Hodgkin's lymphoma (NHL).

Purpose of the Study:

  • To review current literature on the role of HCV in the pathogenesis of NHL.
  • To present a meta-analysis of epidemiological data investigating the association between HCV and NHL development.
  • To discuss therapeutic strategies for HCV-associated NHL, including antiviral therapy and chemotherapy.

Main Methods:

  • Comprehensive literature search for recent studies on HCV and NHL.
  • Meta-analysis of epidemiological data to quantify the risk of NHL in HCV-infected individuals.
  • Review of treatment outcomes for HCV-associated NHL.

Main Results:

  • Evidence confirms the significant role of HCV in the pathogenesis of NHL.
  • Antiviral therapy has demonstrated effectiveness in treating HCV-associated NHL.
  • Chemotherapy remains an option for managing HCV-associated NHL.

Conclusions:

  • HCV infection is a critical factor in the development of NHL.
  • Antiviral treatment is a viable and effective therapeutic option for HCV-associated NHL.
  • Further research into the mechanisms and optimal management of HCV-associated NHL is warranted.

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