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Strength training attenuates post-infarct cardiac dysfunction and remodeling.

Michael A Garza1, Emily A Wason1, Justin R Cruger1

  • 1Laboratory of Cardiovascular Research, Department of Health, Kinesiology, and Nutrition, University of Texas at San Antonio, 1 UTSA Circle, San Antonio, TX, 78249, USA.

The Journal of Physiological Sciences : JPS
|March 27, 2019
PubMed
Summary

Strength training (St) may improve cardiac function after myocardial infarction (MI). Unlike endurance training, St attenuates left ventricle (LV) dilation and enhances cardiac performance, offering a potential therapeutic benefit for post-MI recovery.

Keywords:
Cardiac functionMyocardial infarctionRatsStrength training

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Area of Science:

  • Cardiovascular Physiology
  • Exercise Science
  • Cardiac Rehabilitation

Background:

  • Exercise post-myocardial infarction (MI) aims to improve cardiac function.
  • Endurance training (Et) preserves cardiac function but may increase left ventricle (LV) dimensions.
  • Strength training (St) promotes cardiac hypertrophy and function improvement without dilation.

Purpose of the Study:

  • To investigate the effects of strength training (St) on cardiac function and remodeling in rats post-MI.
  • To compare St effects against a sedentary control group in an MI rat model.

Main Methods:

  • Surgically induced MI in rats, followed by assignment to sedentary (MI-Sed) or strength training (MI-St) groups.
  • MI-St group underwent 10 weeks of ladder climbing with weights, starting 1 week post-MI.
  • Echocardiography and in vivo LV hemodynamic analysis were performed to assess cardiac function and dimensions.

Main Results:

  • Strength training (St) significantly shortened LV end-diastolic dimension in MI rats compared to sedentary controls.
  • Peak contraction (+dP/dt max) and relaxation (-dP/dt max) velocities were significantly enhanced by St.
  • Improved fractional shortening (%FS) was observed, indicating enhanced cardiac contractility.

Conclusions:

  • Strength training (St) may be beneficial for post-myocardial infarction (MI) recovery.
  • St effectively attenuates left ventricle (LV) dilation and improves cardiac dysfunction following MI.
  • These findings suggest St as a potential therapeutic strategy for cardiac remodeling and function post-MI.