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[Histometric studies of placental villi in Rh incompatibility]
The delay in maturation of placental terminal villi in cases of Rh-incompatibility has often been described. The terminal villi of sixteen placentas from pregnancies complicated by Rh-incompatibility histometric were studied. Twenty-two placentas of healthy mothers who had born healthy babies after normal pregnancies served as a control group. The severity of the Rh-incompatibility was classified according to the clinical histories of the newborns and the criteria of Liley. The average area of a cross-section of a terminal villus was 3402 microns 2 (control group 2150 micronsoff; the degree of vascularization was calculated to be 6.5% (control group 30.5%). The other villous parameters measured or calculated show that maturation of the placenta is extremely retarded in cases of Rh-incompatibility. This failure in differentiation can not be compensated for by an enlargement of the organ as a whole. It was shown that the changes in the placental villi are closely correlated to the severity of the fetal disease.
The delay in maturation of placental terminal villi in cases of Rh-incompatibility has often been described. The terminal villi of sixteen placentas from pregnancies complicated by Rh-incompatibility histometric were studied. Twenty-two placentas of healthy mothers who had born healthy babies after normal pregnancies served as a control group. The severity of the Rh-incompatibility was classified according to the clinical histories of the newborns and the criteria of Liley. The average area of a cross-section of a terminal villus was 3402 microns 2 (control group 2150 micronsoff; the degree of vascularization was calculated to be 6.5% (control group 30.5%). The other villous parameters measured or calculated show that maturation of the placenta is extremely retarded in cases of Rh-incompatibility. This failure in differentiation can not be compensated for by an enlargement of the organ as a whole. It was shown that the changes in the placental villi are closely correlated to the severity of the fetal disease.