Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Burn Injuries01:22

Burn Injuries

4.3K
Burn injuries occur when the skin and underlying tissues are damaged due to exposure to heat, electricity, chemicals, radiation, or friction. They can vary in severity, from minor superficial burns to severe deep burns that can be life-threatening.
The damage results in the death of skin cells, which can lead to a massive loss of fluid. Dehydration, electrolyte imbalance, and renal and circulatory failure follow, which can be fatal. Burn patients are treated with intravenous fluids to offset...
4.3K
Acute Kidney Injury I: Introduction01:22

Acute Kidney Injury I: Introduction

632
Introduction:Acute Kidney Injury (AKI) describes a swift decrease in kidney function occurring over hours to days, characterized by the kidneys' failure to remove waste products from the bloodstream. This leads to dangerous complications like metabolic acidosis, fluid overload, and electrolyte imbalances, such as hyperkalemia, which can cause life-threatening arrhythmias. AKI is common in both hospital and outpatient settings, often triggered by dehydration, sepsis, or exposure to nephrotoxic...
632
Acute Kidney Injury II: Pathophysiology01:29

Acute Kidney Injury II: Pathophysiology

962
Acute kidney injury (AKI) causes are categorized into three primary categories based on the location of the injury: prerenal, intrarenal (or intrinsic), and postrenal causes. This classification guides clinical management and illustrates how different pathways can impair kidney function.Etiology and Pathophysiology of Acute Kidney Injury1. Prerenal causesEtiology: Prerenal Acute Kidney Injury, the most common type, occurs when reduced blood flow to the kidneys decreases filtration capacity...
962
Acute Kidney Injury VI: Nursing Management01:22

Acute Kidney Injury VI: Nursing Management

418
Acute Kidney Injury (AKI) results in an inability to maintain fluid, electrolyte, and acid-base balance. Effective nursing management is critical in improving patient outcomes and includes comprehensive patient assessment and targeted interventions.Comprehensive Patient AssessmentA detailed history collection is essential, focusing on any recent infections, nephrotoxic medication use, or chronic conditions such as hypertension and diabetes that may contribute to AKI. During the physical...
418
Acute Kidney Injury V: Interprofessional Care01:20

Acute Kidney Injury V: Interprofessional Care

307
Acute Kidney Injury (AKI) requires a collaborative healthcare approach to restore renal function and prevent complications. Essential management strategies involve monitoring fluid and electrolyte balance, adjusting medications, initiating dialysis when necessary, and providing nutritional support.Fluid and Electrolyte ManagementFluid Monitoring: Regularly monitoring body weight, central venous pressure, and urine output helps detect fluid imbalances early. Patient intake and output are...
307
Pathophysiology of Peptic Ulcer Disease: Injurious Factors01:22

Pathophysiology of Peptic Ulcer Disease: Injurious Factors

1.1K
Peptic ulcers are sores on the stomach's inner lining and the upper small intestine, which are the result of disruptions in the mucosal layer that houses parietal cells which produce gastric acid, and chief cells which secrete pepsinogen.
In the antrum region, G cells secrete the gastrin hormone that binds to gastrin-cholecystokinin-B (CCK2) receptors on parietal and enterochromaffin-like (ECL) cells in the fundic glands. Simultaneously, the vagus nerve releases acetylcholine, which binds...
1.1K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Characteristics of auditory and vestibular symptoms in patients with Meniere's disease.

Brazilian journal of otorhinolaryngology·2026
Same author

Botulinum Toxin Effects and Its Association with Vitamin and Mineral Supplementation: A Narrative Review.

Nutrients·2026
Same author

Chronic Maxillary Sinusitis Due to Material Compatible with Hyaluronic Filler-A Case Report.

Clinics and practice·2025
Same author

Completeness of reporting in abstracts of randomized controlled trials in dental medicine published from 2015-2023: A methodological study.

PloS one·2025
Same author

Potential Role of <i>Malassezia restricta</i> in Pterygium Development.

International journal of molecular sciences·2025
Same author

Pedicled Radial Forearm "Free" Flap for Intraoral Reconstruction Based on an Unexpectedly High Origin of the Radial Artery-Case Report.

Clinics and practice·2025

Related Experiment Video

Updated: Jan 23, 2026

Construction of Cyclic Cell-Penetrating Peptides for Enhanced Penetration of Biological Barriers
10:12

Construction of Cyclic Cell-Penetrating Peptides for Enhanced Penetration of Biological Barriers

Published on: September 19, 2022

2.8K

PENETRATING ORBITOCRANIAL INJURY.

Marisa Klančnik1, Petar Ivanišević1, Slaven Lupi-Ferandin1

  • 11University of Split, Split University Hospital Center, Department of Otorhinolaryngology, Split, Croatia; 2University of Split, Split University Hospital Center, Department of Maxillofacial Surgery, Split, Croatia; 3University of Split, Split University Hospital Center, Department of Neurosurgery, Split, Croatia; 4University of Split, Split University Hospital Center, Department of Ophthalmology, Split, Croatia; 5University of Split, Split University Hospital Center, Department of Nuclear Medicine, Split, Croatia.

Acta Clinica Croatica
|June 7, 2019
PubMed
Summary

A penetrating orbital brain injury caused by a metal rod led to a Bacillus cereus abscess. Prompt surgical repair and targeted antibiotics successfully treated the brain abscess, resulting in full recovery without neurological deficits.

Keywords:
Brain edemaCase reportsEye injuries, penetratingHead injuries, penetratingOrbit

More Related Videos

Controlled Cortical Impact Model for Traumatic Brain Injury
05:30

Controlled Cortical Impact Model for Traumatic Brain Injury

Published on: August 5, 2014

29.6K
Shifting Zebrafish Lethal Skeletal Mutant Penetrance by Progeny Testing
08:39

Shifting Zebrafish Lethal Skeletal Mutant Penetrance by Progeny Testing

Published on: September 1, 2017

8.1K

Related Experiment Videos

Last Updated: Jan 23, 2026

Construction of Cyclic Cell-Penetrating Peptides for Enhanced Penetration of Biological Barriers
10:12

Construction of Cyclic Cell-Penetrating Peptides for Enhanced Penetration of Biological Barriers

Published on: September 19, 2022

2.8K
Controlled Cortical Impact Model for Traumatic Brain Injury
05:30

Controlled Cortical Impact Model for Traumatic Brain Injury

Published on: August 5, 2014

29.6K
Shifting Zebrafish Lethal Skeletal Mutant Penetrance by Progeny Testing
08:39

Shifting Zebrafish Lethal Skeletal Mutant Penetrance by Progeny Testing

Published on: September 1, 2017

8.1K

Area of Science:

  • Neurosurgery
  • Ophthalmology
  • Infectious Diseases

Background:

  • Penetrating orbital injuries are rare but can lead to severe complications.
  • Bacillus cereus is an opportunistic pathogen that can cause serious infections.

Observation:

  • A 37-year-old female sustained a right orbital stab injury with frontal brain lobe penetration from a metal rod.
  • Imaging revealed orbital roof fracture and intracranial bone fragments; Bacillus cereus was isolated from the wound.

Findings:

  • Initial surgical repair involved orbital roof reconstruction and cranioplasty.
  • Postoperative brain abscess developed, treated with intravenous antibiotics (ciprofloxacin, metronidazole) and corticosteroids.
  • Complete abscess regression was observed after 14 weeks of therapy.

Implications:

  • This case highlights the importance of a comprehensive surgical and infectious disease approach for penetrating orbital brain injuries.
  • Successful management of Bacillus cereus brain abscesses can be achieved with timely intervention and appropriate antibiotic therapy.
  • Aggressive treatment can lead to significant recovery, even in complex cases of traumatic brain injury.