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Defective AMH signaling disrupts GnRH neuron development and function and contributes to hypogonadotropic

Samuel Andrew Malone1,2, Georgios E Papadakis3, Andrea Messina3

  • 1Jean-Pierre Aubert Research Center (JPArc), Laboratory of Development and Plasticity of the Neuroendocrine Brain, Inserm, UMR-S 1172, Lille, France.

Elife
|July 11, 2019
PubMed

Keywords:
AMHGnRHKallmann's syndromecell migrationdevelopmental biologygeneticsgenomicshumanmousereproductionAMH signalingGnRH neuron migrationhypogonadotropic hypogonadismgenetic mutationsneurodevelopment

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