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Liver dysfunction in ovarian hyperstimulation syndrome. A case report.

C E Sueldo1, H M Price, K Bachenberg

  • 1Departments of Obstetrics and Gynecology, University of California, San Francisco.

The Journal of Reproductive Medicine
|April 1, 1988
PubMed
Summary

Severe ovarian hyperstimulation syndrome (OHS) can cause liver dysfunction. This case highlights a rare complication of human menopausal gonadotropin therapy, emphasizing the need for vigilant patient monitoring.

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Area of Science:

  • Reproductive endocrinology and infertility treatments.
  • Clinical management of iatrogenic complications in assisted reproduction.

Background:

  • Human menopausal gonadotropin (hMG) therapy is a common treatment for infertility.
  • Ovarian hyperstimulation syndrome (OHS) is a known risk associated with hMG therapy.
  • OHS management typically focuses on fluid shifts and electrolyte imbalances.

Observation:

  • A severe case of OHS was observed in a patient undergoing hMG therapy.
  • The patient presented with significant liver dysfunction, a previously unreported feature of OHS.
  • Clinical presentation included ascites, pleural effusion, and hemoconcentration, alongside hepatic enzyme elevation.

Findings:

  • This case represents the first documented instance of severe OHS manifesting with prominent liver dysfunction.

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  • The liver dysfunction appeared secondary to the systemic inflammatory response and vascular permeability characteristic of severe OHS.
  • Resolution of OHS symptoms, including liver dysfunction, was achieved with conservative management.

    • Implications:

    • Clinicians should consider liver dysfunction in the differential diagnosis of severe OHS.
    • Enhanced vigilance and monitoring are crucial for patients experiencing severe OHS, particularly for hepatic involvement.
    • Further research may elucidate the precise mechanisms linking OHS and liver injury in assisted reproductive technology.