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Related Experiment Video

Updated: Jan 21, 2026

Atom Probe Tomography Studies on the CuIn,GaSe2 Grain Boundaries
09:51

Atom Probe Tomography Studies on the CuIn,GaSe2 Grain Boundaries

Published on: April 22, 2013

13.3K

Chorea.

Pichet Termsarasab

    Continuum (Minneapolis, Minn.)
    |July 30, 2019
    PubMed
    Summary
    This summary is machine-generated.

    This review details the clinical approach to chorea, emphasizing phenomenologic features for differential diagnosis. It highlights recent findings in genetic and acquired choreas and discusses comprehensive treatment strategies beyond symptom management.

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    Area of Science:

    • Neurology
    • Movement Disorders
    • Genetics

    Background:

    • Chorea is a hyperkinetic movement disorder characterized by involuntary, irregular, and unpredictable movements.
    • Differentiating chorea from other movement disorders requires careful evaluation of phenomenologic features.

    Observation:

    • C9orf72 disease is a common cause of Huntington disease-like symptoms.
    • Anti-IgLON5 disease and Sydenham chorea are notable causes of chorea, with increasing interest in immunotherapies for the latter.
    • Benign hereditary chorea involves mutations in genes such as NKX2-1, ADCY5, GNAO1, and PDE10A.

    Findings:

    • New dopamine-depleting agents offer improved symptomatic treatment for chorea with fewer side effects.
    • Deep brain stimulation is an option for some choreic disorders, but requires consideration of non-chorea features and neurodegenerative aspects.

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  • Ongoing research into genetic interventions for Huntington disease shows promise.

    • Implications:

    • Clinical features are paramount for accurate diagnosis and investigation of chorea.
    • Effective management of choreic disorders necessitates a comprehensive, multidisciplinary approach, not solely focused on chorea.
    • Understanding the genetic underpinnings and diverse etiologies of chorea is crucial for tailored therapeutic strategies.