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Absence of a postantibiotic effect in experimental Pseudomonas endocarditis treated with imipenem, with or without

M T Hessen1, P G Pitsakis, M E Levison

  • 1Department of Medicine, Medical College of Pennsylvania, Philadelphia.

Insights

The in vitro postantibiotic effect (PAE) of imipenem and gentamicin against Pseudomonas aeruginosa did not predict in vivo efficacy in a rat endocarditis model. Bacterial regrowth occurred rapidly once imipenem levels dropped below the minimal inhibitory concentration.

Area of Science:

  • Microbiology
  • Pharmacology
  • Infectious Diseases

Background:

  • The postantibiotic effect (PAE) describes the delay in bacterial growth after antibiotic exposure.
  • Imipenem and gentamicin are used to treat Pseudomonas aeruginosa infections.
  • Understanding in vivo PAE is crucial for predicting antibiotic treatment success.

Purpose of the Study:

  • To evaluate the in vivo postantibiotic effect (PAE) of imipenem, alone and with gentamicin, against Pseudomonas aeruginosa endocarditis in rats.
  • To determine if in vitro PAE predicts in vivo efficacy.

Main Methods:

  • In vitro testing of imipenem and imipenem plus gentamicin against Pseudomonas aeruginosa to determine PAE.
  • Induction of Pseudomonas aeruginosa endocarditis in a rat model.
  • Treatment of infected rats with imipenem alone or in combination with gentamicin.
  • Monitoring bacterial counts and antibiotic levels in vegetations.

Main Results:

  • In vitro, imipenem showed a 3-4 hour PAE, and imipenem plus gentamicin showed a 5-hour PAE.
  • In vivo, rapid bactericidal effect was observed initially, but bacterial counts increased as imipenem levels fell below the minimal inhibitory concentration.
  • Gentamicin levels in vegetations did not prolong the PAE observed in vitro.

Conclusions:

  • The in vitro postantibiotic effect of imipenem, with or without gentamicin, does not reliably predict its presence or efficacy in a rat model of Pseudomonas aeruginosa endocarditis.
  • Antibiotic efficacy in vivo is influenced by factors beyond in vitro PAE, such as drug penetration and sustained therapeutic concentrations.

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