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Case report: Patiromer-induced hypercalcemia.

Michael R Wiederkehr1, Ankit N Mehta1, Michael Emmett1

  • 1Texas A&M Health Science Center, College of Medicine, Baylor University Medical Center at Dallas, Division of Nephrology, Department of Internal Medicine, Dallas, TX, USA.

Clinical Nephrology. Case Studies
|August 15, 2019
PubMed
Summary
This summary is machine-generated.

Patiromer, a new potassium binder, may cause hypercalcemia. This condition, characterized by high calcium levels, resolved upon drug discontinuation and recurred upon re-initiation, indicating a direct link.

Keywords:
hypercalcemiahyperkalemiapatiromerpotassium binding agents

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Area of Science:

  • Nephrology
  • Clinical Pharmacology

Background:

  • Patiromer is a novel potassium-binding polymer approved by the FDA for managing hyperkalemia.
  • This ion exchange resin functions by binding potassium in the gastrointestinal tract.
  • A known mechanism involves the release of calcium ions in exchange for bound potassium ions.

Observation:

  • A case study detailing the occurrence of hypercalcemia following the initiation of patiromer therapy.
  • The patient's serum calcium levels decreased when patiromer was discontinued.
  • Hypercalcemia recurred upon the reintroduction of patiromer, suggesting a causal relationship.

Findings:

  • The study identified a potential adverse effect of patiromer: drug-induced hypercalcemia.
  • The cyclical pattern of calcium level changes correlated directly with patiromer administration and withdrawal.
  • This case highlights the importance of monitoring calcium levels in patients receiving patiromer.

Implications:

  • Clinicians should be aware of the risk of hypercalcemia associated with patiromer use.
  • Monitoring serum calcium levels is recommended for patients initiating or on long-term patiromer therapy.
  • Further research may be warranted to elucidate the incidence and risk factors for patiromer-induced hypercalcemia.