Decreasing time to first shock: Routine application of defibrillation pads in prehospital STEMI

Sarah Felder1, Kristine VanAarsen2, Matthew Davis2,3

  • 1St. Thomas Elgin General Hospital, Emergency Medicine Department, St. Thomas, ON.

CJEM
|August 31, 2019
PubMed

Insights

Routine defibrillation pad application in ST-elevation myocardial infarction (STEMI) patients experiencing out-of-hospital cardiac arrest (OHCA) significantly reduces time to defibrillation. This practice is crucial for improving survival rates in cardiac arrest patients.

Area of Science:

  • Emergency Medicine
  • Cardiology
  • Prehospital Care

Background:

  • Four percent of ST-elevation myocardial infarctions (STEMIs) are complicated by out-of-hospital cardiac arrest (OHCA).
  • Shorter time to initial defibrillation in patients with ventricular fibrillation/tachycardia (VF/VT) arrests is linked to increased favorable neurologic survival.

Purpose of the Study:

  • To determine if routine defibrillation pad application in prehospital STEMI patients decreases time to initial defibrillation for those experiencing OHCA.

Main Methods:

  • A health records review of adult STEMI patients from January 2012 to July 2016.
  • Evaluation of a 'pads-on' protocol implemented pre- and post-study period (January 2012-May 2014 vs. June 2014-July 2016).
  • T-test used to compare mean times to defibrillation between groups.

Main Results:

  • Eleven STEMI patients experienced OHCA during paramedic care.
  • The 'pads-on' protocol group (4 patients) had a mean time to defibrillation of 17.7 seconds.
  • The group with pads applied post-arrest (7 patients) had a mean time to defibrillation of 72.7 seconds, a difference of 55.0 seconds.

Conclusions:

  • Routine defibrillation pad application in STEMI patients who suffer OHCA significantly decreases time to initial defibrillation.
  • This reduction in time to defibrillation is associated with improved favorable neurologic survival outcomes.
Abstract

Related Concept Videos

Shock Wave Application to Cell Cultures05:39

Shock Wave Application to Cell Cultures

Shock waves nowadays are well known for their regenerative effects. Therefore in vitro experiments are of increasing interest. We therefore developed a model for in vitro shock wave trials (IVSWT) that enables us to mimic in vivo conditions thereby avoiding distracting physical...
13.2K
Uncontrolled Hemorrhagic Shock Modeled via Liver Laceration in Mice with Real Time Hemodynamic Monitoring06:11

Uncontrolled Hemorrhagic Shock Modeled via Liver Laceration in Mice with Real Time Hemodynamic Monitoring

Uncontrolled hemorrhage, an important cause of mortality among trauma patients, can be modeled using a standard liver laceration in a murine model. This model results in consistent blood loss, survival, and allows for testing hemostatic agents. This article provides the step-by-step process to perform this valuable...
9.1K
Prehospital Thrombolysis: A Manual from Berlin05:52

Prehospital Thrombolysis: A Manual from Berlin

Identification of suspected stroke in the dispatch center of the Berlin Fire Brigade prompts the deployment of a CT-equipped ambulance. If ischemic stroke is confirmed and contraindications are excluded prehospital thrombolysis is...
22.3K
Application of RNAi and Heat-shock-induced Transcription Factor Expression to Reprogram Germ Cells to Neurons in C. elegans07:53

Application of RNAi and Heat-shock-induced Transcription Factor Expression to Reprogram Germ Cells to Neurons in C. elegans

This protocol describes how to study cellular processes during cell fate conversion in Caenorhabditis elegans in vivo. Using transgenic animals, allowing heat-shock promoter-driven overexpression of the neuron fate-inducing transcription factor CHE-1 and RNAi-mediated depletion of the chromatin-regulating factor LIN-53 germ cell to neuron reprogramming can be observed in vivo.
8.1K
Decreased Body Temperature01:29

Decreased Body Temperature

A decreased body temperature can occur in patients with hypothermia and frostbite. Heat loss with extended cold exposure overpowers the body's ability to create heat, resulting in hypothermia. Core temperature readings help classify hypothermia. Mild hypothermia is temperatures between 32 °C (89.6 °F) and 35°C (95 °F) and is caused by impaired thermoregulation. Moderate hypothermia is temperatures between 28 C (82.4 °F) and 32 °C (89.6 °F) caused by...
1.0K
Decreased pulse rate01:14

Decreased pulse rate

Bradycardia is a medical condition in which the heart rate is slower than normal. It occurs when the heart's natural pacemaker, the sinus node, generates slower electrical impulses than the standard rhythm. In adults, bradycardia is diagnosed when the pulse rate falls below 60 beats per minute, indicating a deviation from the normal heart rate range.
There are specific risk factors that can elevate the likelihood of developing bradycardia. Advanced age is a significant factor, with...
852