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Intestinal lymphoid tissue changes in the cortisone-treated Wistar rat.

E Carpen, A Olinic, M E Gocan

    Morphologie Et Embryologie
    |April 1, 1985
    PubMed
    Summary
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    Cortisone administration initially caused intestinal lymphoid tissue hyperplasia and bone marrow hypercellularity in Wistar rats. Following a 6-week recovery, cellular depletion was observed alongside normalized bone marrow and lymph node parenchyma.

    Area of Science:

    • Endocrinology
    • Immunology
    • Histology

    Background:

    • Previous research investigated lymphoid tissue responses under varied conditions.
    • Endocrine imbalance is known to affect immune system components.

    Purpose of the Study:

    • To examine the morphological changes in intestinal lymphoid tissue of male Wistar rats following cortisone administration.
    • To understand the recovery process of lymphoid tissues after endocrine disruption.

    Main Methods:

    • Male Wistar rats were administered cortisone.
    • Intestinal lymphoid tissue, lymph nodes, and bone marrow were analyzed morphologically at 7 days post-administration and after a 6-week recovery interval.

    Main Results:

    • Cortisone induced intestinal lymphoid tissue hyperplasia, lymph node parenchyma depletion, and bone marrow hypercellularity at 7 days.

    Related Experiment Videos

  • After 6 weeks, lymphoid tissue showed cellular depletion, with restored lymph node parenchyma and normocellular bone marrow.

    • Conclusions:

    • Cortisone administration causes significant, time-dependent alterations in lymphoid tissue morphology.
    • The lymphoid system exhibits a capacity for recovery following short-term endocrine imbalance induced by cortisone.