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Sugar, opioids and binge eating.

D T Fullerton, C J Getto, W J Swift

    Brain Research Bulletin
    |June 1, 1985
    PubMed
    Summary

    Endogenous opiates influence feeding behaviors, especially sugar intake, in animals. Emerging evidence suggests sugar consumption may also impact the human opiate system, potentially linking it to obesity and stress-related eating.

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    Area of Science:

    • Neuroscience
    • Behavioral Science
    • Endocrinology

    Background:

    • Endogenous opiates play a role in regulating feeding behaviors in animal models.
    • Obesity in animals is often linked to altered opiate production and receptor activity.
    • Nutrients, particularly sugars, influence feeding behavior via opiate pathways in rodents.

    Purpose of the Study:

    • To explore the role of endogenous opiates in feeding behavior.
    • To investigate the connection between nutrient intake, particularly sugar, and the opiate system.
    • To examine the potential influence of the opiate system on human eating patterns, obesity, and stress-related feeding.

    Main Methods:

    • Review of existing research on endogenous opiates and feeding in animal models.
    • Analysis of clinical observations regarding carbohydrate intake, binge eating, and obesity in humans.
    • Examination of recent studies on the effects of sugar on beta-endorphin production in obese individuals.

    Main Results:

    • Opiate antagonists block the effects of palatable diets and stress-induced sucrose ingestion in rodents.
    • Clinical data suggests a link between carbohydrate consumption, binge eating, and obesity in humans.
    • Recent findings indicate that sugar intake may increase beta-endorphin levels in obese subjects.

    Conclusions:

    • Endogenous opiates are significantly involved in controlling feeding, especially sugar consumption, in experimental animals.
    • While animal studies are clear, the role of nutrients in the human opiate system requires further investigation.
    • Clinical evidence suggests a potential connection between sugar, the opiate system, and human obesity and stress-related eating behaviors.

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