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When the Heart Cries Wolf: Myocardial Bridging Presenting as Angina-like Chest Pain
Fnu Farukhuddin1, Muhammad Akrmah2, Maryam R Hussain2
1Neurology, University Hospital Cleveland Medical Center, Cleveland, USA.
Insights
Myocardial bridging, a common congenital heart anomaly, can cause angina-like chest pain. This case highlights a rare mid-segment LAD artery compression, emphasizing diagnostic challenges in atypical chest pain presentations.
Area of Science:
- Cardiology
- Congenital Heart Disease
Background:
- Myocardial bridging (MB) is the most common congenital coronary anomaly, characterized by an epicardial coronary artery segment tunneling through the myocardium.
- The left anterior descending artery (LAD) is most frequently affected, often associated with hypertrophic cardiomyopathy (HCM).
Observation:
- A 64-year-old female presented with severe, non-exertional left-sided chest pain.
- Initial electrocardiography (EKG) and echocardiography were unremarkable, but stress EKG revealed significant ST-segment depressions.
Findings:
- Coronary angiography demonstrated systolic compression of the mid-segment LAD artery, with normal caliber during diastole.
- This presentation is atypical due to the mid-segment involvement and severe anginal symptoms.
Implications:
- This case underscores the diagnostic challenges posed by myocardial bridging presenting with atypical chest pain.
- Early recognition and appropriate management, such as beta-blocker therapy, are crucial for patients with symptomatic myocardial bridging.
Abstract:
Myocardial bridging (MB) is the most common congenital coronary anomaly and refers to an intramural course of an epicardial coronary artery. The proximal segment of the left anterior descending artery (LAD) is the most commonly involved vessel and is often seen in patients with hypertrophic cardiomyopathy (HCM). We present a case of a 64-year-old female with left-sided non-exertional chest pain. Electrocardiography (EKG) and echocardiography were negative, however, stress EKG was positive with deep ST-segment depressions. Coronary angiography revealed mid-segment compression of LAD during systole, returning to its normal caliber during diastole. The patient remained asymptomatic during the hospital course and was later discharged on beta-blocker therapy. This case is different from others in a sense that it presented with severe pain like angina and mid-segment of LAD is involved rather than the proximal segment where it commonly occurs. This case report will help clinicians overcome the diagnostic challenge in patients presenting with atypical chest pain.
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