Search research articles

ABOUT JoVE

Overview Leadership Blog JoVE Help Center

AUTHORS

Publishing Process Editorial Board Scope & Policies Peer Review FAQ Submit

LIBRARIANS

Testimonials Subscriptions Access Resources Library Advisory Board FAQ

RESEARCH

JoVE Journal Methods Collections JoVE Encyclopedia of Experiments Archive

EDUCATION

JoVE Core JoVE Business JoVE Science Education JoVE Lab Manual Faculty Resource Center Faculty Site

Terms & Conditions of Use

Related Concept Videos

Local Anesthetics: Differential Sensitivity of Nerve Fibers

Local Anesthetics: Differential Sensitivity of Nerve Fibers

Local anesthetics (LAs) block the sodium channels of nerve trunks, sensory nerve endings, and neuromuscular junctions. Although LAs can block all kinds of nerves, the sensitivity of nerve fibers differs according to nerve types and structures. LAs are known to block myelinated fibers faster than unmyelinated ones. Also, they block pain or sensory neurons at low concentrations without affecting the motor neurons involved in muscle contractions. This helps relieve labor pain without affecting the...

Sympathetic Pathways: Sympathetic Chain Ganglia

Sympathetic Pathways: Sympathetic Chain Ganglia

The sympathetic chain ganglia, also known as the sympathetic trunk ganglia or paravertebral ganglia, are a series of ganglia located bilaterally on either side of the spinal column. These ganglia serve as relay stations for the sympathetic nervous system. Preganglionic neurons originating in the spinal cord project their axons to the sympathetic chain ganglia. Within the ganglia, these preganglionic fibers synapse with postganglionic neurons.The postganglionic neurons of the sympathetic trunk...

Major Somatic Sensory Pathways

Major Somatic Sensory Pathways

Sensory impulses related to touch, pressure, vibration, and proprioception from various body parts, such as the limbs, trunk, neck, and posterior head, travel to the cerebral cortex through the posterior column-medial lemniscus pathway. The pathway’s name derives from the two white-matter tracts that convey the impulses: the spinal cord's posterior column and the brainstem's medial lemniscus. First-order sensory neurons extend their axons into the spinal cord, forming the...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by

Same author

Nonmotor Symptoms in Amyotrophic Lateral Sclerosis and Their Correlation With Disease Progression.

Journal of clinical neuromuscular disease·2021

Same author

Characteristics of Guillain-Barré syndrome in a Lebanese tertiary care center.

Neuromuscular disorders : NMD·2021

Same author

Traumatic Brain Injury: Oxidative Stress and Novel Anti-Oxidants Such as Mitoquinone and Edaravone.

Antioxidants (Basel, Switzerland)·2020

Same author

Characteristics of amyotrophic lateral sclerosis in Lebanon-a chart review.

Amyotrophic lateral sclerosis & frontotemporal degeneration·2020

Same author

Guillain-Barre Syndrome.

Seminars in neurology·2019

Same author

Adult onset Sandhoff disease: a rare mimicker of amyotrophic lateral sclerosis.

Amyotrophic lateral sclerosis & frontotemporal degeneration·2019

Same journal

Isolated Cranial Tremors: A Reappraisal.

Seminars in neurology·2026

Same journal

Dystonia and Tremor.

Seminars in neurology·2026

Same journal

Pendular nystagmus and oculopalatal tremor.

Seminars in neurology·2026

Same journal

Sanjay Pandey, MBBS, MD, DNB, DM, FIAN, and Aasef Shaikh, MD, PhD.

Seminars in neurology·2026

Same journal

Tremor.

Seminars in neurology·2026

Same journal

Carotid Disease.

Seminars in neurology·2026

See all related articles

Search research articles

Related Experiment Video

Updated: Jan 5, 2026

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1

Published on: February 13, 2018

Common Entrapment Neuropathies.

Elia Malek¹, Johnny S Salameh¹

¹Department of Neurology, American University of Beirut Medical Center, Riad El Solh, Beirut, Lebanon.

Seminars in Neurology

|October 23, 2019

Summary

This summary is machine-generated.

Entrapment neuropathies involve peripheral nerve compression. Early diagnosis using anatomy, symptoms, and tests like electrophysiology and ultrasonography is key for effective treatment plans.

More Related Videos

A Simple Approach to Induce Experimental Autoimmune Neuritis in C57BL/6 Mice for Functional and Neuropathological Assessments

A Simple Approach to Induce Experimental Autoimmune Neuritis in C57BL/6 Mice for Functional and Neuropathological Assessments

Published on: November 9, 2017

The Sciatic Nerve Cuffing Model of Neuropathic Pain in Mice

The Sciatic Nerve Cuffing Model of Neuropathic Pain in Mice

Published on: July 16, 2014

Related Experiment Videos

Last Updated: Jan 5, 2026

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1

Published on: February 13, 2018

A Simple Approach to Induce Experimental Autoimmune Neuritis in C57BL/6 Mice for Functional and Neuropathological Assessments

A Simple Approach to Induce Experimental Autoimmune Neuritis in C57BL/6 Mice for Functional and Neuropathological Assessments

Published on: November 9, 2017

The Sciatic Nerve Cuffing Model of Neuropathic Pain in Mice

The Sciatic Nerve Cuffing Model of Neuropathic Pain in Mice

Published on: July 16, 2014

Area of Science:

Neurology
Orthopedic Surgery
Anatomy

Background:

Entrapment neuropathies are common conditions involving peripheral nerve compression.
Their varied presentations necessitate specialized knowledge for accurate diagnosis and treatment.
Understanding topographic anatomy and clinical signs is crucial.

Purpose of the Study:

To provide a comprehensive overview of common entrapment neuropathies.
To highlight the importance of diagnostic tools in clinical practice.
To guide the development of effective treatment strategies.

Main Methods:

Review of common entrapment neuropathies in the upper and lower extremities.
Emphasis on detailed topographic anatomy and clinical manifestations.
Integration of electrophysiological studies and neuromuscular ultrasonography.

Main Results:

Discussion of the most frequent forms of nerve entrapment.
Highlighting the diagnostic utility of electrophysiology and ultrasonography.
Underscoring the need for accurate diagnosis for patient management.

Conclusions:

Accurate diagnosis of entrapment neuropathies relies on comprehensive knowledge and appropriate diagnostic tools.
Effective management requires understanding anatomy, clinical presentation, and electrodiagnostic findings.
This review aids neurologists and orthopedic surgeons in diagnosing and treating these conditions.