Idiopathic Hypertrophic Pyloric Stenosis with Complete Ladd's Band: A Rare Association

Ahmed M Abo Elyazeed1, Mohamed M Shalaby1, Mohamed M Awad1

  • 1Division of Surgery, Department of Pediatric Surgery, Tanat University Hospital, Tanta, Gharbia, Egypt.

Insights

This case report highlights a rare instance of concurrent idiopathic hypertrophic pyloric stenosis and intestinal malrotation in an infant. Early laparoscopic exploration is crucial for accurate diagnosis and effective treatment of complex pediatric surgical conditions.

Area of Science:

  • Pediatric Surgery
  • Gastrointestinal Surgery
  • Neonatal Care

Background:

  • Idiopathic hypertrophic pyloric stenosis (IHPS) is a common cause of infant vomiting.
  • Intestinal malrotation with Ladd's bands is a serious congenital anomaly requiring prompt surgical intervention.
  • Concurrent presentation of IHPS and malrotation is exceptionally rare, posing diagnostic challenges.

Observation:

  • A 45-day-old male infant presented with persistent projectile nonbilious vomiting.
  • Initial ultrasound suggested IHPS, leading to laparoscopic pyloromyotomy.
  • Postoperative vomiting persisted, necessitating further investigation.

Findings:

  • Laparoscopic re-exploration confirmed a complete pyloromyotomy but revealed coexisting intestinal malrotation with a complete Ladd's band.
  • Conversion to open laparotomy and performance of the Ladd's procedure resolved the vomiting.
  • Histopathological confirmation of IHPS was obtained.

Implications:

  • This case underscores the importance of thorough laparoscopic exploration in persistent postoperative vomiting, even after initial corrective surgery.
  • It highlights the rarity and diagnostic complexity of concurrent IHPS and malrotation.
  • A systematic diagnostic approach, including exploration, is vital for managing complex neonatal surgical emergencies.

Related Concept Videos

Peptic Ulcer Disease I: Introduction01:30

Peptic Ulcer Disease I: Introduction

Peptic Ulcer Disease (PUD) is characterized by mucosal excavation in the esophagus, stomach, pylorus, or duodenum. It can manifest as acute or chronic based on the extent and duration of mucosal involvement.
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
694
Peptic Ulcer Disease V: Surgical Management and Nursing Care01:25

Peptic Ulcer Disease V: Surgical Management and Nursing Care

Surgical management and nursing care are crucial in treating Peptic Ulcer Disease (PUD). Here is an organized and enhanced overview of the surgical interventions and the associated nursing care for PUD:
Surgical Interventions for Peptic Ulcer Disease
768
Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies01:28

Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies

Peptic ulcer disease (PUD) presents with diverse symptoms depending on the location and severity of the ulcer. Clinical manifestations of peptic ulcer include dull pain and a burning sensation in the mid-epigastric region.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
495
Pathophysiology of Peptic Ulcer Disease: Injurious Factors01:22

Pathophysiology of Peptic Ulcer Disease: Injurious Factors

Peptic ulcers are sores on the stomach's inner lining and the upper small intestine, which are the result of disruptions in the mucosal layer that houses parietal cells which produce gastric acid, and chief cells which secrete pepsinogen.
In the antrum region, G cells secrete the gastrin hormone that binds to gastrin-cholecystokinin-B (CCK2) receptors on parietal and enterochromaffin-like (ECL) cells in the fundic glands. Simultaneously, the vagus nerve releases acetylcholine, which binds...
1.0K
Esophageal Strictures-II: Clinical Features and Management01:26

Esophageal Strictures-II: Clinical Features and Management

Patients with esophageal strictures often experience a range of symptoms. Initially, they may have difficulty swallowing solid foods, which can progress to include liquids. Additional symptoms may involve chest pain or discomfort, regurgitating food and fluids, heartburn, unintentional weight loss, coughing or choking during meals, and hoarseness.
Healthcare providers should gather a comprehensive medical history and conduct a physical examination for diagnosis. If esophageal stricture is...
367
Mitral Stenosis I: Introduction01:22

Mitral Stenosis I: Introduction

Mitral Valve Stenosis (MVS) is a heart condition where the mitral valve narrows, impeding blood circulation from the left atrium to the left ventricle. The etiology and pathophysiology of this condition are multifaceted, leading to a cascade of cardiovascular complications.Causes of Mitral Valve StenosisRheumatic Heart Disease: It is the main cause of mitral valve stenosis, particularly in developing nations. This condition arises from rheumatic fever, an inflammatory illness resulting from...
350