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Pre- and Postoperative Care for Interventional Skin Rejuvenation
Kaete A Archer1, Paul J Carniol2
1Aesthetic Institute of Manhattan for Facial and Plastic Surgery, 460 Park Avenue, 17th Floor, New York, NY 10022, USA.
Facial Plastic Surgery Clinics of North America
|November 30, 2019
Summary
Facial aging involves tissue descent, fat atrophy, bone changes, and skin aging. Photoaging, distinct from genetic aging, is caused by UV radiation-induced free radicals damaging skin cells.
Area of Science:
- Dermatology and aging research.
Background:
- Facial aging results from multiple factors including tissue descent, fat atrophy, bone remodeling, and skin changes.
- Photoaging, or skin aging due to light exposure, is a distinct process from intrinsic, genetically determined aging.
- UV radiation exposure generates free radicals, initiating cellular damage.
Purpose of the Study:
- To differentiate the mechanisms of photoaging from intrinsic facial aging.
- To elucidate the cellular damage pathways involved in photoaging.
Main Methods:
- Histological and clinical examination of skin aging.
- Analysis of cellular damage markers.
Main Results:
- Photoaging presents distinct clinical and histological features compared to intrinsic aging.
- UV-A and UV-B radiation induce free radicals, leading to DNA mutations.
- Free radicals also cause structural and enzymatic protein alterations and lipid peroxidation in the skin.
Conclusions:
- Photoaging is a significant contributor to facial aging, driven by UV-induced oxidative stress.
- Understanding photoaging mechanisms is crucial for developing targeted interventions against skin aging.
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