The Apoptosis Regulator 14-3-3η and Its Potential as a Therapeutic Target in Pituitary Oncocytoma
Sida Zhao1, Bin Li2, Chuzhong Li1
1Cell Biology, Beijing Neurosurgical Institute, Capital Medical University, Beijing, China.
Abstract:
The 14-3-3 protein family has attracted much attention in research into the pathogenesis of human tumors because of its involvement in tumorigenesis. In previous studies, we found that 14-3-3η was highly expressed in pituitary oncocytoma. However, the mechanism by which 14-3-3η regulates tumorigenesis in pituitary oncocytoma is unclear. 14-3-3η-binding proteins were investigated in pituitary oncocytoma by immunoprecipitation and proteomic analysis. A total of 443 proteins were identified as 14-3-3η binding proteins. The interactions of 14-3-3η and its binding partners were identified by a network analysis using the STRING database. The network included 433 nodes and 564 edges. PRAS40 (AKT1S1) was a binding protein of 14-3-3η and showed experimental interactions with 14-3-3η in the STRING database. The combined score was 0.407, which suggested a functional link. The 443 binding proteins of 14-3-3η showed enriched molecular signatures in GSEA and GO analysis. PRAS40 (AKT1S1) was enriched in the mTOR signaling pathway. Western blot analysis showed that the relative expression of p-PRAS40 (T246)/PRAS40 was significantly higher in pituitary oncocytoma than in normal pituitary tissues (p < 0.05). R18, a 14-3-3 protein inhibitor, inhibited MMQ cell proliferation after treatment with 8 μM R18 for 48 h compared to the control group (p < 0.01). These results suggest that 14-3-3η may be involved in promoting tumorigenesis in pituitary oncocytoma by interacting with PRAS40 (T246) via the mTOR signaling pathway.
Insights
The 14-3-3η protein promotes pituitary oncocytoma growth by interacting with PRAS40 (AKT1S1) in the mTOR pathway. Inhibiting 14-3-3η reduces tumor cell proliferation, suggesting a therapeutic target.
Area of Science:
- Molecular Biology
- Oncology
- Biochemistry
Background:
- The 14-3-3 protein family is implicated in human tumor pathogenesis.
- 14-3-3η is highly expressed in pituitary oncocytoma, but its role in tumorigenesis is unclear.
- Understanding 14-3-3η's mechanism in pituitary oncocytoma is crucial for therapeutic development.
Purpose of the Study:
- To investigate the mechanism of 14-3-3η in pituitary oncocytoma tumorigenesis.
- To identify 14-3-3η binding proteins and their functional interactions.
- To explore the role of PRAS40 (AKT1S1) and the mTOR signaling pathway.
Main Methods:
- Immunoprecipitation and proteomic analysis to identify 14-3-3η binding proteins.
- Network analysis using STRING database to map protein interactions.
- Gene Set Enrichment Analysis (GSEA) and Gene Ontology (GO) analysis.
- Western blot to assess protein expression levels.
- Cell proliferation assays using a 14-3-3 protein inhibitor (R18).
Main Results:
- Identified 443 14-3-3η binding proteins, including PRAS40 (AKT1S1).
- PRAS40 (AKT1S1) was enriched in the mTOR signaling pathway.
- Phosphorylated PRAS40 (T246) levels were significantly higher in pituitary oncocytoma.
- Inhibition of 14-3-3η reduced pituitary tumor cell proliferation.
Conclusions:
- 14-3-3η promotes pituitary oncocytoma tumorigenesis through interaction with PRAS40 (AKT1S1).
- The mTOR signaling pathway is involved in this interaction.
- 14-3-3η represents a potential therapeutic target for pituitary oncocytoma.
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