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Updated: Jan 1, 2026

A Doxorubicin-induced Cardiomyopathy Model in Adult Zebrafish
Published on: June 7, 2018
LongShengZhi capsule inhibits doxorubicin-induced heart failure by anti-oxidative stress
Shuai Xu1, Yuanyu Wang1, Maoyun Yu2
1Key Laboratory of Metabolism and Regulation for Major Diseases of Anhui Higher Education Institutes, College of Food and Biological Engineering, Hefei University of Technology, Hefei, China.
Insights
LongShengZhi capsule (LSZ) effectively treats doxorubicin-induced heart failure in mice. LSZ reduces cardiac fibrosis, inflammation, and apoptosis, offering a potential therapeutic for heart failure.
Area of Science:
- Cardiology
- Pharmacology
- Traditional Chinese Medicine
Background:
- Heart failure is a significant global health burden.
- Doxorubicin (DOX) is a chemotherapy agent known to induce cardiotoxicity.
- Traditional Chinese Medicine (TCM) offers potential therapeutic avenues for cardiovascular diseases.
Purpose of the Study:
- To investigate the cardioprotective effects of LongShengZhi capsule (LSZ) against doxorubicin (DOX)-induced heart failure in a mouse model.
- To elucidate the underlying mechanisms of LSZ's therapeutic action.
Main Methods:
- C57BL/6 mice were divided into control, DOX-induced heart failure, and DOX + LSZ treatment groups.
- DOX was administered to induce heart failure, while LSZ was given orally with chow.
- Cardiac function markers, fibrosis, apoptosis, inflammation, oxidative stress, and relevant gene/protein expressions were assessed.
Main Results:
- LSZ treatment significantly ameliorated DOX-induced increases in serum cardiac enzymes and cardiac fibrosis.
- LSZ inhibited DOX-induced apoptosis by reducing caspase 3 activity and expression.
- LSZ decreased inflammatory cytokine levels and reduced oxidative stress by upregulating antioxidant enzymes (SOD1, SOD2, catalase, GPx1) via FOXO3A and SIRT3 activation.
Conclusions:
- LSZ demonstrates significant cardioprotective effects against DOX-induced heart failure in mice.
- LSZ mitigates heart failure by reducing oxidative stress, inflammation, and apoptosis.
- LSZ holds promise as a potential therapeutic agent for heart failure management.
Abstract:
Heart failure is a major cause of morbidity and mortality worldwide. LongShengZhi capsule (LSZ), a traditional Chinese medicine, is used for treatment of patients with vascular diseases. Herein we investigated the effect of LSZ treatment on doxorubicin (DOX)-induced heart failure in mice. C57BL/6 mice randomly in 3 groups received following treatment: Control group, mice were fed normal chow; DOX group, mice were intraperitoneally injected DOX to induce heart failure and fed normal chow; and LSZ group, mice were injected DOX and fed normal chow containing LSZ. DOX induced heart failure as evidenced by increased serum creatine kinase, lactic dehydrogenase and α-hydroxybutyrate dehydrogenase, and cardiac fibrosis. However, LSZ treatment substantially inhibited DOX-induced heart failure parameters. Mechanistically, LSZ reduced collagen content and fibrosis by inhibiting expression of collagen type I α1 (COL1α1), COL1α2, α-smooth muscle actin and transforming growth factor β1. In addition, DOX-induced cell apoptosis was inhibited by LSZ, coupled with reduced caspase 3 activity and mRNA expression. LSZ decreased inflammatory cytokine levels. More importantly, LSZ decreased oxidative stress by inducing expression of anti-oxidative stress enzymes including superoxide dismutase 1 (SOD1), SOD2, catalase and glutathione peroxidase 1 through activation of forkhead box O3A and sirtuin 3. In conclusion, our study demonstrates that LSZ reduces heart failure by reducing production of reactive oxygen species and inhibiting inflammation/apoptosis. Our study also suggests the potential application of LSZ for heart failure treatment.
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