Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Glaucoma: Overview01:25

Glaucoma: Overview

1.2K
Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
1.2K
Angle Closure Glaucoma: Treatment01:28

Angle Closure Glaucoma: Treatment

1.1K
Angle-closure glaucoma, or closed-angle glaucoma, is an eye condition where the iris bulges out and blocks the iridocorneal angle, resulting in a buildup of aqueous humor and increased intraocular pressure. Immediate medical attention is necessary due to the sudden onset of symptoms. The treatment for angle-closure glaucoma includes short-term and long-term approaches. Short-term treatment involves using eye drops like pilocarpine to lower intraocular pressure by increasing aqueous humor...
1.1K
Open Angle Glaucoma: Treatment01:27

Open Angle Glaucoma: Treatment

905
In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
Drugs such as carbonic anhydrase inhibitors, α2- and...
905
Photoreceptors and Visual Pathways01:22

Photoreceptors and Visual Pathways

8.5K
At the molecular level, visual signals trigger transformations in photopigment molecules, resulting in changes in the photoreceptor cell's membrane potential. The photon's energy level is denoted by its wavelength, with each specific wavelength of visible light associated with a distinct color. The spectral range of visible light, classified as electromagnetic radiation, spans from 380 to 720 nm. Electromagnetic radiation wavelengths exceeding 720 nm fall under the infrared category,...
8.5K
Adrenal Gland Disorders01:27

Adrenal Gland Disorders

2.8K
Adrenal gland disorders manifest when the production of adrenal hormones deviates from the norm, resulting in either excessive or insufficient concentrations.
Adrenal insufficiency, characterized by insufficient cortisol and aldosterone production, leads to conditions like Addison's disease. This disorder, affecting the adrenal cortex, exhibits symptoms such as skin bronzing, dehydration, low blood pressure, fatigue, and weight loss. Congenital adrenal hyperplasia, a genetic ailment causing...
2.8K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Clinical impact of a single time-point protocol paired FDG-PET and radioactive iodine scan for comprehensive disease assessment in differentiated thyroid cancer.

Current problems in cancer·2026
Same author

Glucagon-like peptide-1 receptor (GLP-1R) overexpression defines a distinct immunogenetic subset in primary and metastatic thyroid cancer: implications for GLP-1R agonist therapy.

Frontiers in oncology·2026
Same author

Anti-Obesity Effects of <i>Tisochrysis lutea</i> Powder in High-Fat Diet-Induced Obese Mice Through the Regulation of Adipogenesis and Lipid Metabolism.

International journal of molecular sciences·2026
Same author

From discovery to therapeutic development of monoclonal antibodies.

Journal of controlled release : official journal of the Controlled Release Society·2026
Same author

Neuroendocrine and neural control of bone mass in health and disease.

The Journal of clinical investigation·2026
Same author

Calculating Glucagon-Like Pepide-1 Receptor Agonist-Associated Medullary Thyroid Cancer Risk: A Novel Integration of the Surveillance, Epidemiology and End Results Cancer Registry and the FDA Adverse Event Reporting System.

Head & neck·2026
Same journal

Sulfasalazine-Induced Hypoglycemia in a Patient with Type 2 Diabetes and End-Stage Renal Disease.

AACE clinical case reports·2025
Same journal

SDHD Gene Mutations: Looking Beyond Head and Neck Tumors.

AACE clinical case reports·2025
Same journal

Mild Thyrotoxicosis due to Seronegative Graves' Disease or Disseminated Thyroid Autonomy.

AACE clinical case reports·2025
Same journal

Improvement of Glycogenic Hepatopathy With Minimal Corresponding Improvement of Glycemic Control in a Person With Type 1 Diabetes: Case Report and Literature Review.

AACE clinical case reports·2025
Same journal

Insulinoma: A Novel Presentation of Multiple Endocrine Neoplasia 4.

AACE clinical case reports·2025
Same journal

A Novel Mutation in a Family With Multiple Endocrine Neoplasia Type 1 and Aggressive Pancreatic Neuroendocrine Tumors.

AACE clinical case reports·2025
See all related articles

Related Experiment Video

Updated: Dec 30, 2025

Full-Circle Cauterization of Limbal Vascular Plexus for Surgically Induced Glaucoma in Rodents
10:10

Full-Circle Cauterization of Limbal Vascular Plexus for Surgically Induced Glaucoma in Rodents

Published on: February 15, 2022

1.8K

CUSHING DISEASE MASQUERADING AS GLAUCOMA.

Yumiko Tsushima, Lubna Bashir Munshi, Charit Taneja

    AACE Clinical Case Reports
    |January 23, 2020
    PubMed
    Summary
    This summary is machine-generated.

    Glaucoma can be an early sign of Cushing disease, a condition of excess cortisol. Promptly treating this endogenous hypercortisolism can reverse glaucoma and prevent vision loss.

    More Related Videos

    Glaucoma-inducing Procedure in an In Vivo Rat Model and Whole-mount Retina Preparation
    08:30

    Glaucoma-inducing Procedure in an In Vivo Rat Model and Whole-mount Retina Preparation

    Published on: March 12, 2016

    13.5K
    Ultrasound Cyclo Plasty in Eyes with Glaucoma
    05:05

    Ultrasound Cyclo Plasty in Eyes with Glaucoma

    Published on: January 26, 2018

    12.6K

    Related Experiment Videos

    Last Updated: Dec 30, 2025

    Full-Circle Cauterization of Limbal Vascular Plexus for Surgically Induced Glaucoma in Rodents
    10:10

    Full-Circle Cauterization of Limbal Vascular Plexus for Surgically Induced Glaucoma in Rodents

    Published on: February 15, 2022

    1.8K
    Glaucoma-inducing Procedure in an In Vivo Rat Model and Whole-mount Retina Preparation
    08:30

    Glaucoma-inducing Procedure in an In Vivo Rat Model and Whole-mount Retina Preparation

    Published on: March 12, 2016

    13.5K
    Ultrasound Cyclo Plasty in Eyes with Glaucoma
    05:05

    Ultrasound Cyclo Plasty in Eyes with Glaucoma

    Published on: January 26, 2018

    12.6K

    Area of Science:

    • Endocrinology
    • Ophthalmology

    Background:

    • Glaucoma is a known side effect of exogenous corticosteroids.
    • Glaucoma from endogenous corticosteroid overproduction is rare, with few reported cases.
    • This study highlights glaucoma as a potential manifestation of endogenous hypercortisolism.

    Observation:

    • A case of glaucoma presenting initially in a patient with Cushing disease (CD) is reported.
    • The patient had a cushingoid appearance and elevated cortisol levels.
    • Diagnostic tests confirmed CD, and pituitary imaging suggested a small adenoma.

    Findings:

    • The patient underwent transsphenoidal resection for an adrenocorticotropic hormone-positive adenoma.
    • Post-surgery, the patient's hypercortisolism and intraocular pressures improved.
    • This suggests a direct link between endogenous hypercortisolism and glaucoma development.

    Implications:

    • Glaucoma can lead to irreversible blindness if not managed.
    • Treatment of underlying Cushing disease can reverse glaucoma caused by endogenous hypercortisolism.
    • Increased awareness of extraocular manifestations of secondary glaucoma is crucial for timely diagnosis and treatment.