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Identifying multi-hit carcinogenic gene combinations: Scaling up a weighted set cover algorithm using compressed

Qais Al Hajri1, Sajal Dash2, Wu-Chun Feng1,2

  • 1Department of Electrical and Computer Engineering, Virginia Tech, Blacksburg, VA, 24060, USA.

Scientific Reports
|February 8, 2020
PubMed

Insights

Researchers developed a faster algorithm to identify multi-hit gene combinations in cancer, improving diagnosis and paving the way for targeted therapies. This computational advance aids in understanding cancer

Area of Science:

  • Computational biology
  • Genomics
  • Cancer research

Background:

  • Effective cancer treatments are limited due to the complex genetic mutations underlying different cancer instances.
  • Identifying combinations of mutated genes (multi-hit combinations) is crucial for understanding cancer etiology.
  • Previous algorithms were computationally intensive, limiting the analysis to two-hit combinations.

Purpose of the Study:

  • To enhance an algorithm for identifying multi-hit gene combinations, specifically three or more genetic mutations.
  • To improve computational efficiency for analyzing complex cancer mutation data.
  • To enable the identification of potential driver mutations for targeted cancer therapies.

Main Methods:

  • Developed a compressed binary matrix representation for genetic data.
  • Optimized the algorithm for parallel processing on a graphics processing unit (GPU).
  • Compared the GPU implementation's speed against a CPU-based implementation.

Main Results:

  • The GPU-optimized algorithm achieved an estimated 12,144-fold speed increase for 3-hit combination identification.
  • Identified 3-hit gene combinations differentiated tumor from normal samples with 90% sensitivity and 93% specificity.
  • Demonstrated the potential to identify driver mutations from mutation distribution patterns.

Conclusions:

  • The enhanced GPU algorithm efficiently identifies complex multi-hit gene combinations in cancer.
  • These findings offer insights into cancer origins and support the development of targeted combination therapies.
  • Further experimental validation is recommended to confirm driver mutations and therapeutic strategies.

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