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[Problems and difficulties in adapting period in infants and children after massive intestinal resection]

S Hasegawa1, Y Hirai, Y Sanada

  • 1Department of Pediatric Surgery, Juntendo University School of Medicine, Tokyo, Japan.

Nihon Geka Gakkai Zasshi
|September 1, 1988
PubMed

Insights

Children with massive intestinal resection face long-term nutritional challenges, including hepatic dysfunction and abnormal vitamin levels, requiring extended nutritional support. This highlights the need for careful monitoring and management post-surgery.

Area of Science:

  • Pediatric Surgery
  • Gastroenterology
  • Nutritional Science

Context:

  • Massive intestinal resection in infants and children leads to significant intestinal adaptation challenges.
  • Study included 17 survivors of 31 pediatric patients who underwent massive intestinal resection (residual intestine < 75 cm) between 1966-1987.

Purpose:

  • To define the difficulties and problems encountered during intestinal adaptation after massive intestinal resection in pediatric patients.
  • To investigate the long-term nutritional sequelae in survivors of massive intestinal resection.

Summary:

  • Postoperative hepatic dysfunction was observed in 11 of 17 survivors, with reduced ileal length potentially contributing to its recurrence.
  • Disturbances in bile acid reabsorption and rapid turnover persisted even after catch-up growth.
  • Abnormal serum mineral levels during total parenteral nutrition (TPN) normalized with oral feeding, while vitamin deficiencies (Vitamins A, E, D3, B12) persisted into school age.
  • Massive intestinal resection necessitates long-term nutritional care, particularly for lipid and vitamin management.

Impact:

  • Identifies persistent nutritional deficiencies and hepatic dysfunction as key challenges following pediatric massive intestinal resection.
  • Emphasizes the critical need for prolonged, specialized nutritional support, including lipid and vitamin supplementation, for affected children.
  • Provides insights into the etiology of hepatic dysfunction related to ileal length and bile acid metabolism.

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