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Structural and functional analysis of mitochondrial plasmids in Claviceps purpurea

A Düvell1, H Hessberg-Stutzke, B Oeser

  • 1Institut für Mikrobiologie, Universität Düsseldorf, Federal Republic of Germany.

Molecular & General Genetics : MGG
|September 1, 1988
PubMed

Insights

Claviceps purpurea mitochondrial plasmids are structurally related and potentially mobile genetic elements. Their replication and transcription suggest viral origins, with no significant phenotypic impact observed in plasmid-free strains.

Area of Science:

  • Mycology
  • Molecular Biology
  • Genetics

Background:

  • Claviceps purpurea, a phytopathogenic Ascomycete, harbors mitochondrial (mt) plasmids.
  • These plasmids exist in high molar excess compared to mitochondrial DNA (mtDNA).

Purpose of the Study:

  • To investigate the structural relationships and potential mobility of C. purpurea mt plasmids.
  • To analyze the replication and transcription of a specific plasmid (pClK1).
  • To assess the phenotypic impact of plasmid presence or absence.

Main Methods:

  • Comparative analysis of plasmids from four geographically distinct strains.
  • Restriction mapping and nucleotide sequencing of plasmid pClK1.
  • Analysis of plasmid transcripts via Northern blotting.
  • Phenotypic comparison of plasmid-containing and plasmid-free strains.

Main Results:

  • All analyzed plasmids were structurally related, featuring terminal inverted repeats (TIRs), suggesting possible mobility.
  • Plasmid pClK1 exhibits a perfect 327 bp TIR and its sequence indicates an adenovirus-like replication mechanism.
  • pClK1 is highly transcribed into two major mRNAs (3.5 and 3.15 kb), which are absent in plasmid-free strains.
  • Curing strains of plasmids did not result in significant phenotypic differences.

Conclusions:

  • C. purpurea mt plasmids are structurally conserved and may be mobile genetic elements.
  • The replication and transcription patterns of pClK1 suggest viral ancestry.
  • The functional significance of these plasmids remains unclear as their absence does not alter the host phenotype.

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