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Published on: December 9, 2016
Drug-Induced Resistance in Micrometastases: Analysis of Spatio-Temporal Cell Lineages
Judith Pérez-Velázquez1, Katarzyna A Rejniak2,3
1Mathematical Modeling of Biological Systems, Centre for Mathematical Science, Technical University of Munich, Garching, Germany.
Abstract:
Resistance to anti-cancer drugs is a major cause of treatment failure. While several intracellular mechanisms of resistance have been postulated, the role of extrinsic factors in the development of resistance in individual tumor cells is still not fully understood. Here we used a hybrid agent-based model to investigate how sensitive tumor cells develop drug resistance in the heterogeneous tumor microenvironment. We characterized the spatio-temporal evolution of lineages of the resistant cells and examined how resistance at the single-cell level contributes to the overall tumor resistance. We also developed new methods to track tumor cell adaptation, to trace cell viability trajectories and to examine the three-dimensional spatio-temporal lineage trees. Our findings indicate that drug-induced resistance can result from cells adaptation to the changes in drug distribution. Two modes of cell adaptation were identified that coincide with microenvironmental niches-areas sheltered by cell micro-communities (protectorates) or regions with limited drug penetration (refuga or sanctuaries). We also recognized that certain cells gave rise to lineages of resistant cells (precursors of resistance) and pinpointed three temporal periods and spatial locations at which such cells emerged. This supports the hypothesis that tumor micrometastases do not need to harbor cell populations with pre-existing resistance, but that individual tumor cells can adapt and develop resistance induced by the drug during the treatment.
Insights
Individual tumor cells can develop drug resistance through adaptation to the tumor microenvironment, not necessarily from pre-existing resistant populations. This resistance emerges during treatment, influenced by drug distribution and cellular niches.
Area of Science:
- Oncology
- Computational Biology
- Cancer Research
Background:
- Anti-cancer drug resistance is a primary reason for treatment failure.
- Intracellular mechanisms of resistance are known, but extrinsic factors remain less understood.
- The tumor microenvironment's role in developing individual cell drug resistance requires further investigation.
Purpose of the Study:
- To investigate how sensitive tumor cells acquire drug resistance within a heterogeneous tumor microenvironment.
- To characterize the spatio-temporal evolution of resistant cell lineages.
- To understand how single-cell resistance contributes to overall tumor resistance.
Main Methods:
- Utilized a hybrid agent-based model to simulate tumor cell behavior.
- Developed novel methods for tracking tumor cell adaptation and viability.
- Analyzed three-dimensional spatio-temporal lineage trees of resistant cells.
Main Results:
- Drug-induced resistance can arise from cellular adaptation to altered drug distribution.
- Identified two adaptation modes linked to microenvironmental niches: protectorates and refuga/sanctuaries.
- Discovered specific cells (precursors of resistance) that generate resistant lineages at distinct temporal and spatial points.
Conclusions:
- Tumor micrometastases may not require pre-existing resistant cells to develop resistance.
- Individual tumor cells can adapt and develop drug resistance during therapy.
- The tumor microenvironment significantly influences the emergence and spread of drug resistance.
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