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Miconazole and amphotericin B alter polymorphonuclear leukocyte functions and membrane fluidity in similar fashions

K Yasui1, M Masuda, T Matsuoka

  • 1Department of Pediatrics, Shinshu University School of Medicine, Matsumoto, Japan.

Insights

Miconazole and amphotericin B (AmB) impair polymorphonuclear leukocytes (PMN) function by altering their membrane properties. These antifungal drugs reduce PMN migration and adherence, impacting immune cell behavior.

Area of Science:

  • Immunology
  • Pharmacology
  • Cell Biology

Background:

  • Polymorphonuclear leukocytes (PMNs) are crucial immune cells.
  • Antifungal agents like miconazole and amphotericin B (AmB) can affect immune cell function.
  • Understanding drug effects on PMNs is vital for managing infections.

Purpose of the Study:

  • To investigate and compare the effects of miconazole and amphotericin B (AmB) on human polymorphonuclear leukocytes (PMNs).
  • To assess how these antifungal drugs influence PMN chemotaxis, adherence, and membrane properties.

Main Methods:

  • Human PMNs were preincubated with miconazole or AmB at therapeutic concentrations.
  • Chemotaxis was assessed using N-formylmethionyl-leucyl-phenylalanine (FMLP).
  • Membrane fluidity was measured using an excimer-forming lipid technique, and adherence was quantified via a nylon fiber column method.

Main Results:

  • Miconazole and AmB pretreatment irreversibly reduced PMN random migration and chemotaxis.
  • Both drugs increased PMN adherence.
  • Miconazole and AmB decreased PMN membrane fluidity and lowered FMLP receptor binding affinity.

Conclusions:

  • Miconazole and AmB significantly alter key in vitro membrane properties of human PMNs.
  • These alterations in PMN function may have implications for host immune response during antifungal therapy.
  • The study highlights the immunomodulatory effects of common antifungal medications.

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