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Left ventricular structure and function, assessed by imaging and Doppler echocardiography, in athletes engaged in

C Van den Broeke1, R Fagard

  • 1Department of Pathophysiology, Faculty of Medicine, University of Leuven, Belgium.

Insights

Strength training in male athletes did not alter cardiac structure or left ventricular (LV) function. Echocardiography revealed no significant differences in LV dimensions, mass, or systolic/diastolic function between throwers and controls.

Area of Science:

  • Cardiology
  • Sports Medicine
  • Exercise Physiology

Background:

  • Athletes often exhibit cardiac adaptations due to intense training.
  • The specific impact of throwing events on left ventricular (LV) structure and function requires further investigation.

Purpose of the Study:

  • To assess cardiac structure and LV systolic and diastolic function in male athletes participating in throwing events compared to matched controls.
  • To determine if strength training in these athletes influences LV diastolic function.

Main Methods:

  • Echocardiography and Doppler velocimetry were used to evaluate cardiac parameters in ten male throwers and ten age, height, and weight-matched controls.
  • Assessment included LV internal diameter, wall thickness, LV mass, systolic function, early diastolic function (relaxation velocity, inflow pattern), and late diastolic function.

Main Results:

  • No significant differences were found in LV internal diameter, wall thickness, LV mass, or systolic LV function between athletes and controls.
  • Both early and late LV diastolic function, including LV relaxation and filling patterns, were similar in throwers and non-athletes.
  • The ratio of peak filling velocities during atrial contraction and early filling remained unchanged, indicating unaltered LV distensibility.

Conclusions:

  • The training regimen of these male throwing athletes was not associated with alterations in LV structure or function.
  • Strength training, as performed by these athletes, does not appear to induce detrimental changes in cardiac morphology or hemodynamics.

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