Related Concept Videos
The Tumor Microenvironment
Metastasis
Epithelial-to-Mesenchymal Transition
The epithelial-to-mesenchymal transition or EMT is a developmental process commonly observed in wound healing, embryogenesis, and cancer metastasis. EMT is induced by transforming growth factor-beta (TGF-β) or receptor tyrosine kinase (RTK) ligands, which further...
Cancer Stem Cells and Tumor Maintenance
Cancer stem cells are thought to originate from tissue-specific normal stem cells or progenitor cells. The normal stem cells usually reside in...
Drugs that Stabilize Microtubules
Stem Cell Niche
Tumor Immunotherapy
You might also read
Related Articles
Articles linked to this work by shared authors, journal, and citation graph.
Related Experiment Video
Updated: Dec 17, 2025

Studying the Effects of Tumor-Secreted Paracrine Ligands on Macrophage Activation using Co-Culture with Permeable Membrane Supports
Published on: November 28, 2019
Stellate Cells in the Tumor Microenvironment.
David Roife1,2, Bhaswati Sarcar2, Jason B Fleming3
1Department of Surgery, University of South Florida Morsani College of Medicine, Tampa, FL, USA.
Stellate cells, crucial for wound repair, also drive liver and pancreatic cancer progression. Targeting these cells in the tumor microenvironment offers new therapeutic strategies for these cancers.
More Related Videos
09:52A Mimic of the Tumor Microenvironment: A Simple Method for Generating Enriched Cell Populations and Investigating Intercellular Communication
Published on: September 20, 2016
09:18Heteromulticellular Stromal Cells in Scaffold-free 3D Cultures of Epithelial Cancer Cells to Drive Invasion
Published on: April 4, 2025
Area of Science:
- Oncology
- Cell Biology
- Pathology
Background:
- Tumor microenvironments resemble chronic wounds, implicating wound-repair cells in cancer.
- Stellate cells, found in the liver and pancreas, support wound healing via growth factor and extracellular matrix secretion.
- Dysregulated wound healing involving stellate cells can lead to fibrosis, such as liver cirrhosis.
Purpose of the Study:
- To review the structure and function of hepatic and pancreatic stellate cells.
- To explore the role of stellate cells in the tumor microenvironment of liver and pancreatic cancers.
- To identify potential new therapeutic targets based on stellate cell functions.
Main Methods:
- Literature review and synthesis of existing research on stellate cells.
- Analysis of stellate cell contributions to carcinogenesis and tumor progression.
- Discussion of therapeutic strategies targeting stellate cells.
Main Results:
- Stellate cells contribute to fibrosis, liver cirrhosis, and hepatocellular carcinoma.
- These cells promote tumor growth, invasion, metastasis, immune evasion, and chemotherapy resistance in liver and pancreas.
- Stellate cells are vital components of the tumor stroma in these organs.
Conclusions:
- Hepatic and pancreatic stellate cells play significant roles in cancer development and progression.
- Understanding stellate cell biology is key to developing novel cancer therapies.
- Targeting stellate cells presents a promising avenue for treating liver and pancreatic cancers.