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A Porcine Model of Acute Autologous Pulmonary Embolism

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Published on: September 6, 2024

COVID-19: A Potential Risk Factor for Acute Pulmonary Embolism.

Angel I Martin¹, Gaurav Rao¹

¹NORTHWELL HEALTH, NORTH SHORE UNIVERSITY HOSPITAL, MANHASSET, NEW YORK.

Methodist Debakey Cardiovascular Journal

|July 17, 2020

Summary

COVID-19 infection may increase the risk of acute pulmonary embolisms (APEs). Further research is needed to confirm if COVID-19 is an independent risk factor for APEs, especially with high inflammatory markers.

Keywords:

acute pulmonary embolism acute respiratory distress syndrome computed tomography angiogram coronavirus venous thromboembolism

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Lung CT Segmentation to Identify Consolidations and Ground Glass Areas for Quantitative Assesment of SARS-CoV Pneumonia

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Area of Science:

Cardiology
Pulmonology
Infectious Diseases

Background:

COVID-19 is a novel infectious disease with a wide range of clinical manifestations.
Venous thromboembolic events (VTEs) have been anecdotally reported in COVID-19 patients.
Elevated inflammatory markers are common in severe COVID-19 cases.

Observation:

A COVID-19-positive patient presented with pleuritic chest pain and cough.
Diagnostic imaging revealed acute pulmonary embolisms (APEs) in the patient.
This case aligns with increasing observational reports of VTEs in COVID-19 patients.

Findings:

The patient's presentation suggests a potential link between COVID-19 and APEs.
Elevated inflammatory markers may be associated with an increased risk of VTEs in COVID-19.
COVID-19 may be an independent risk factor for APEs.

Implications:

Further investigation is warranted to establish COVID-19 as an independent risk factor for APEs.
Understanding this association can inform clinical management and risk stratification for COVID-19 patients.
This highlights the importance of considering VTEs in the differential diagnosis of respiratory symptoms in COVID-19 patients.