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Differential DNase I sensitivity of the two complementary nucleosomal DNA strands in cycloheximide-treated Ehrlich

C C Chipev1, V S Ivanova, R Tsanev

  • 1Institute of Molecular Biology, Bulgarian Academy of Sciences, Sofia.

Insights

Newly replicated DNA strands in Ehrlich ascites tumor (EAT) cells show asymmetric accessibility. Parental DNA fragments are more accessible near nucleosomes, suggesting chromatin interactions influence strand exposure.

Area of Science:

  • Molecular Biology
  • Chromatin Structure
  • DNA Replication

Background:

  • Understanding DNA strand accessibility is crucial for DNA replication and repair.
  • Chromatin structure, including nucleosome organization, influences DNA accessibility.
  • Ehrlich ascites tumor (EAT) cells provide a model for studying rapidly dividing cells.

Purpose of the Study:

  • To investigate the accessibility of complementary DNA strands in newly replicated chromatin.
  • To determine if chromatin structure affects the accessibility of parental DNA strands during replication.

Main Methods:

  • EAT cells were cultured with cycloheximide to inhibit protein synthesis.
  • DNA was labeled with 14C-thymidine (bulk) and bromodeoxyuridine/3H-thymidine (new strands).
  • DNase I digestion of isolated nuclei followed by CsCl density gradient centrifugation was used to isolate newly replicated DNA.

Main Results:

  • A dense fraction containing 15-20% newly replicated DNA was obtained.
  • 14C-labeled parental DNA fragments complementary to 3H-nascent strands were predominantly found in the 30-50 nucleotide range.
  • Simulation experiments suggest enhanced accessibility of parental strands at the nucleosomal 5'-end region.

Conclusions:

  • Newly replicated DNA strands exhibit asymmetric accessibility within chromatin.
  • The H2a-H2b dimer interaction site at the nucleosomal 5'-end of parental DNA may be more accessible.
  • Chromatin-induced interactions likely contribute to this observed strand accessibility asymmetry.

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