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Erratum: Long Noncoding RNA (lncRNA) Maternally Expressed Gene 3 (MEG3) Participates in Chronic Obstructive Pulmonary
Hui Bi1, Gui Wang2, Zhiying Li1
1Department of Respiratory Medicine, The Third Affiliated Hospital of Soochow University, Changzhou, Jiangsu, China (mainland).
Abstract:
Figure 3 was incorrectly published in the article titled Long Noncoding RNA (lncRNA) Maternally-Expressed Gene 3 (MEG3) Participates in Chronic Obstructive Pulmonary Disease Through Regulating Human Pulmonary Microvascular Endothelial Cell Apoptosis; PMID: 32201430; PMCID: PMC7111098; DOI: 10.12659/MSM.920793. The correct Figure 3 is as follows.
Insights
This correction clarifies Figure 3 in research on Long Noncoding RNA (lncRNA) Maternally-Expressed Gene 3 (MEG3) and its role in Chronic Obstructive Pulmonary Disease (COPD) pathogenesis. The corrected figure is essential for understanding endothelial cell apoptosis regulation in COPD.
Area of Science:
- Pulmonary Medicine
- Molecular Biology
- Cellular Biology
Background:
- Chronic Obstructive Pulmonary Disease (COPD) is a major global health concern.
- Long Noncoding RNA (lncRNA) Maternally-Expressed Gene 3 (MEG3) has emerged as a key regulator in various cellular processes.
- Endothelial cell apoptosis plays a critical role in COPD pathogenesis.
Purpose of the Study:
- To provide the corrected Figure 3 for the article investigating the role of lncRNA MEG3 in COPD.
- To ensure accurate representation of data regarding human pulmonary microvascular endothelial cell apoptosis.
- To facilitate a correct understanding of MEG3's mechanism in COPD.
Main Methods:
- The study involved the analysis and correction of Figure 3.
- The research focuses on molecular and cellular mechanisms in pulmonary endothelial cells.
- Data pertains to apoptosis regulation influenced by lncRNA MEG3.
Main Results:
- The original Figure 3 was found to be incorrectly published.
- The corrected Figure 3 accurately depicts the relationship between MEG3 and endothelial cell apoptosis in COPD.
- This correction is vital for the interpretation of the study's findings.
Conclusions:
- Accurate data presentation is crucial for scientific integrity.
- The corrected Figure 3 supports the conclusion that MEG3 regulates endothelial cell apoptosis in COPD.
- This research contributes to understanding the molecular underpinnings of COPD.
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