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Related Concept Videos

Amnesia01:13

Amnesia

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Amnesia is a condition marked by long-term memory loss, which impairs the ability to recall past events or create new memories.
The severity and duration of memory loss vary depending on the type and underlying cause. Amnesia is classified into two main types: retrograde and anterograde.
Retrograde amnesia is marked by the loss of memories formed before the onset of the condition. Patients may recall distant past events but often forget those occurring shortly before the incident.
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Traumatic Memory01:20

Traumatic Memory

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Emotionally traumatic events often lead to memories that are exceptionally vivid and enduring, sometimes persisting with remarkable clarity throughout an individual's life. A classic example of this phenomenon is a person who survives a car accident. Even years later, they may recall every detail of the event with startling accuracy — the screeching of the tires, the jarring impact, and the acrid smell of burning rubber. Such vividness contrasts sharply with how an individual...
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Higher Mental Functions of Brain: Learning and Memory01:26

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Memory is one of the most vital higher mental functions of the brain. Memory is closely related to learning because it enables us to retain information and experiences from our past to use them in our present life. It also helps us to remember facts, events, and skills, such as riding a bike or swimming. There are two types of memory — declarative memory, which involves memorizing facts or events, and procedural memory, which enables us to remember how to do something like writing or...
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Post-traumatic Stress Disorder

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Post-traumatic stress disorder (PTSD) is a psychiatric condition that arises following exposure to traumatic events such as natural disasters, forced displacement, or severe accidents. It significantly impairs individuals' ability to cope with daily activities and disrupts their emotional and psychological equilibrium.
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Dissociative Amnesia01:21

Dissociative Amnesia

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Dissociative amnesia is a complex psychological condition that manifests as an inability to recall personal information, often tied to traumatic or stressful events. Unlike general amnesia, individuals with this condition retain the ability to perform routine activities and procedural tasks, such as operating a phone or navigating public transportation, yet experience profound gaps in autobiographical memory. These lapses may encompass significant life events, such as suicide attempts or...
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Long-term depression, or LTD, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTD is the process of synaptic weakening that occurs over time between pre and postsynaptic neuronal connections. The synaptic weakening of LTD works in opposition to synaptic strengthening by long-term potentiation (LTP) and together are the main mechanisms that underlie learning and memory.
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Assessing Changes in Synaptic Plasticity Using an Awake Closed-Head Injury Model of Mild Traumatic Brain Injury
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Post-concussion Vulnerability to Transient Global Amnesia.

Matthew D Garvey1, Caitlin J Miller1, Esther U Kim1

  • 1Department of Neurology, Hospital for Special Surgery, New York, NY, United States.

Frontiers in Neurology
|December 2, 2020
PubMed
Summary
This summary is machine-generated.

Transient global amnesia (TGA) can occur after a concussion. Following TGA, concussion symptoms worsened, suggesting a prolonged period of brain vulnerability post-injury.

Keywords:
amnesiacerebral blood flow (CBF)cerebral vulnerabilityconcussionmTBI (mild traumatic brain injury)transient global amnesia (TGA)

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Area of Science:

  • Neurology
  • Neuroscience
  • Traumatic Brain Injury

Background:

  • Transient global amnesia (TGA) is a distinct neurological condition.
  • The relationship between TGA and concussion sequelae is not well-understood.
  • Concussion recovery can be complex and prolonged.

Observation:

  • A 43-year-old male experienced TGA 22 days post-concussion.
  • Neurological imaging (CT, MRI) and EEG were normal.
  • Full cognitive recovery from TGA occurred within 8 hours.

Findings:

  • Post-TGA, the patient reported significant worsening of concussion symptoms.
  • Symptoms included headache, fogginess, fatigue, and memory difficulties.
  • The patient remained symptomatic for 32 days after the TGA episode.

Implications:

  • A prolonged window of cerebral vulnerability may exist after concussion.
  • This vulnerability could increase susceptibility to acute cognitive events like TGA.
  • Further research is needed to understand TGA in the context of TBI recovery.