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Rat jejunal disaccharidase activity increases biphasically during early post-natal development

E K Lund1, M W Smith

  • 1A.F.R.C. Institute of Animal Physiology and Genetics Research, Babraham, Cambridge.

Insights

Hydrocortisone injection in young rats accelerates sucrase appearance in the jejunum, mimicking normal development. This suggests complex control mechanisms for alpha-glucosidase induction during development.

Area of Science:

  • Biochemistry
  • Developmental Biology
  • Gastroenterology

Background:

  • Disaccharidase development in the rat jejunum is crucial for nutrient absorption.
  • Steroid hormones are thought to play a role in mimicking developmental changes at weaning.

Purpose of the Study:

  • To investigate the effect of hydrocortisone injection on jejunal disaccharidase appearance in young rats.
  • To compare the enzyme induction patterns with normal developmental processes.

Main Methods:

  • Injection of hydrocortisone into 9-day-old rats.
  • Cytochemical analysis of disaccharidase (alpha-glucosidase) activity in jejunal enterocytes.
  • Measurement of enterocyte migration rates and crypt depth.

Main Results:

  • Hydrocortisone accelerated the appearance of sucrase and other alpha-glucosidases, with similar time courses to normal development.
  • Enzyme activity initiated at the villus base and spread upwards.
  • Enterocyte migration rates were similar between hydrocortisone-treated and early normal development phases.
  • The rate of alpha-glucosidase increase in enterocytes was similar after hydrocortisone treatment and in early normal development, but increased significantly later in normal development.

Conclusions:

  • Hydrocortisone injection can precociously induce jejunal disaccharidase activity in rats.
  • The regulation of alpha-glucosidase induction during normal development is more complex than previously assumed.
  • Cell migration rates differ between early and late phases of normal development, suggesting distinct regulatory mechanisms.

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