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Bilateral traumatic third nerve palsy.

F Tognetti1, U Godano, E Galassi

  • 1Division of Neurosurgery, Ospedale Bellaria, Bologna, Italy.

Surgical Neurology
|February 1, 1988
PubMed
Summary
This summary is machine-generated.

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Bilateral third cranial nerve palsy resulted from a head injury, causing paralysis of voluntary and autonomic functions. A midline mesencephalic lesion was identified as the cause in this head-injured patient.

Area of Science:

  • Neuroscience
  • Neurology
  • Ophthalmology

Background:

  • Third cranial nerve palsy can arise from various causes, including trauma.
  • Bilateral involvement is less common but significant, affecting both voluntary and autonomic functions.

Observation:

  • A case study of a head-injured patient presenting with bilateral third cranial nerve palsy.
  • Delayed computed tomography revealed a midline necrotic lesion in the mesencephalon.

Findings:

  • The mesencephalic lesion, likely from a focal contusion, impacted both third nerve nuclear complexes.
  • This resulted in paralysis of voluntary and autonomic functions of the third cranial nerve.
  • Gaze mechanisms and long tracts showed less severe damage compared to the nuclear complexes.

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Implications:

  • This case highlights a specific mechanism of bilateral third nerve palsy following head trauma.
  • Understanding such lesions is crucial for diagnosing and managing traumatic brain injuries with ocular motor deficits.
  • Further research into mesencephalic lesions and their impact on cranial nerves is warranted.