Clinical Review: Navitoclax as a Pro-Apoptotic and Anti-Fibrotic Agent

Nur Najmi Mohamad Anuar1, Nur Syahidah Nor Hisam1, Sze Ling Liew1

  • 1Programme of Biomedical Science, Centre for Toxicology & Health Risk Studies, Faculty of Health Sciences, Universiti Kebangsaan Malaysia, Kuala Lumpur, Malaysia.

Frontiers in Pharmacology
|December 31, 2020
PubMed

Insights

Navitoclax, a BCL-2 inhibitor, targets anti-apoptotic proteins to trigger programmed cell death. Clinical studies show its efficacy in various cancers and fibrotic diseases, with manageable side effects like low platelet count.

Area of Science:

  • Molecular Biology
  • Oncology
  • Pharmacology

Background:

  • B-cell lymphoma 2 (BCL-2) family proteins regulate programmed cell death, with anti-apoptotic and pro-apoptotic members.
  • Inhibitors targeting anti-apoptotic BCL-2 proteins augment the intrinsic apoptotic pathway, offering therapeutic potential.

Purpose of the Study:

  • To elucidate the pro-apoptotic mechanisms of Navitoclax.
  • To review early and current clinical studies of Navitoclax as a single agent and in combination therapy.
  • To present future prospects for Navitoclax clinical development.

Main Methods:

  • Review of existing literature on BCL-2 family proteins and Navitoclax.
  • Analysis of clinical trial data (Phase I and II) for Navitoclax efficacy and safety.
  • Examination of Navitoclax's mechanism of action and drug interactions.

Main Results:

  • Navitoclax exhibits high affinity for anti-apoptotic BCL-2 proteins (BCL-2, BCL-W, BCL-XL).
  • Navitoclax demonstrates efficacy as a single agent in small cell lung cancer and acute lymphocytic leukemia.
  • Combination therapy with Navitoclax enhances chemotherapeutic effects in solid tumors; thrombocytopenia is a known side effect.

Conclusions:

  • Navitoclax is a promising BCL-2 inhibitor with broad therapeutic applications in oncology and fibrotic diseases.
  • Its efficacy is influenced by BCL-2 family member expression.
  • Further clinical development is warranted to optimize its therapeutic use.

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