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Caffeine, mental stress, and risk for hypertension: a cross-cultural replication

L Greenstadt1, L Yang, D Shapiro

  • 1Department of Psychiatry and Biohavioral Sciences, University of California, Los Angeles 90024.

Psychosomatic Medicine
|January 1, 1988
PubMed

Insights

Caffeine and mental stress increase systolic blood pressure, but caffeine does not worsen stress effects. A family history of hypertension amplifies blood pressure responses to caffeine and stress.

Area of Science:

  • Cardiovascular Physiology
  • Human Physiology
  • Pharmacology

Background:

  • Hypertension is a significant global health concern.
  • Understanding factors influencing blood pressure reactivity is crucial for prevention.
  • Caffeine and psychological stress are common modulators of cardiovascular function.

Purpose of the Study:

  • To investigate the independent and combined effects of caffeine and mental arithmetic stress on systolic blood pressure (SBP) and heart rate (HR).
  • To examine whether a family history of hypertension influences cardiovascular responses to caffeine and stress.

Main Methods:

  • Double-blind, placebo-controlled study involving 36 Chinese nationals.
  • Participants received 0, 125, or 250 mg of caffeine across three sessions.
  • SBP and HR were measured following caffeine administration and mental arithmetic stress, with a 12-hour caffeine abstention period prior to each session.

Main Results:

  • Both caffeine and mental stress independently elevated SBP.
  • Caffeine did not potentiate the SBP response to mental stress.
  • No significant effect of caffeine on HR was observed.
  • Individuals with a family history of hypertension showed greater SBP increases with higher caffeine doses and during initial mental stress exposure.

Conclusions:

  • Caffeine and mental stress are independent stressors affecting SBP.
  • Individuals with a family history of hypertension exhibit heightened cardiovascular reactivity to caffeine and acute stress.
  • These findings reinforce the concept of increased cardiovascular risk in genetically predisposed individuals.

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