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Glucose metabolism involved in PD-L1-mediated immune escape in the malignant kidney tumour microenvironment
Yongbo Yu1,2, Ye Liang2, Dan Li2
1Department of Urology, The Affiliated Hospital of Qingdao University, Qingdao, 266003, China.
Abstract:
Programmed death receptor-ligand 1 (PD-L1) plays a crucial role in immune evasion by tumour cells. Most tumour cells exhibit energy dependency and acquire energy from glycolysis. However, the relationship between glucose metabolism and PD-L1 expression remains unclear. In this study, changes in PD-L1 expression in renal carcinoma cells were evaluated during glucose deficiency and recovery, and PD-L1 could inversely regulate glycolysis. In addition, the possible signalling pathways activated by a low level of glucose to regulate PD-L1 were tested experimentally. The results showed that glucose deficiency could upregulate PD-L1 expression in two renal cancer cell lines, 786-O and OS-RC-2. Although the native levels of PD-L1 differed in the two cell lines, the upregulated PD-L1 expression was repristinated after glucose recovery. Moreover, epidermal growth factor receptor (EGFR) expression was upregulated in both cell lines with glucose deficiency. The use of an EGFR inhibitor reversed the upregulation of PD-L1 expression induced by glucose deficiency and inhibited the phosphorylation of extracellular regulated protein kinases 1 and 2 (ERK1/2). EGFR activated by epidermal growth factor (EGF) induced PD-L1 expression and ERK1/2 phosphorylation. Furthermore, an ERK1/2 inhibitor inhibited the phosphorylation of c-Jun and decreased the elevated PD-L1 expression induced by glucose deficiency. In addition, this study also showed that 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase 3 (PFK-2/FBPase 3 or PFKFB3) mediated upregulation of the level of glycolysis to improve the adverse environment through PD-L1 induction. Therefore, glucose metabolism can regulate the expression of PD-L1 through the EGFR/ERK/c-Jun pathway in renal cancer, and elevated PD-L1 can also regulate glycolysis by improving the expression of PFKFB3. The findings of this study could provide a new multiple target treatment for renal cell carcinoma (RCC) therapy.
Insights
Glucose deficiency increases PD-L1 expression in renal cancer cells via the EGFR/ERK/c-Jun pathway. This elevated PD-L1 then enhances glycolysis, suggesting a novel therapeutic target for renal cell carcinoma (RCC).
Area of Science:
- Cancer Biology
- Metabolic Regulation
- Immunotherapy
Background:
- Programmed death receptor-ligand 1 (PD-L1) is vital for tumor immune evasion.
- Tumor cells rely on glycolysis for energy, but its link to PD-L1 is unclear.
- Understanding this relationship may reveal new therapeutic strategies for renal cell carcinoma (RCC).
Purpose of the Study:
- To investigate the impact of glucose metabolism on PD-L1 expression in renal carcinoma.
- To elucidate the signaling pathways involved in glucose-regulated PD-L1.
- To explore the reciprocal regulation between PD-L1 and glycolysis in RCC.
Main Methods:
- Assessed PD-L1 and epidermal growth factor receptor (EGFR) expression in 786-O and OS-RC-2 renal cancer cells under glucose deficiency and recovery.
- Utilized EGFR and extracellular regulated protein kinases 1 and 2 (ERK1/2) inhibitors to probe signaling pathways.
- Investigated the role of 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase 3 (PFKFB3) in mediating glycolysis.
Main Results:
- Glucose deficiency upregulated PD-L1 expression in both renal cancer cell lines.
- EGFR signaling, through the EGFR/ERK/c-Jun pathway, mediated glucose-deficiency-induced PD-L1 upregulation.
- Elevated PD-L1 expression promoted glycolysis by upregulating PFKFB3.
Conclusions:
- Glucose metabolism critically influences PD-L1 expression in renal cancer via the EGFR/ERK/c-Jun axis.
- PD-L1 reciprocally regulates glycolysis, enhancing it under adverse conditions.
- The interplay between glucose metabolism and PD-L1 presents a promising multi-target therapeutic avenue for RCC.
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