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Blood flow in the upper limb during brachial plexus anaesthesia
1Department of Anaesthesia, Wessex Centre for Plastic and Maxillofacial Surgery and Burns, Odstock Hospital, Salisbury, Wiltshire.
Anaesthesia
|April 1, 1988
Summary
Brachial plexus anesthesia with lignocaine and adrenaline increased cardiac output and finger blood flow. Adrenaline likely caused vasoconstriction at the injection site, impacting blood flow distribution.
Area of Science:
- Anesthesiology
- Cardiovascular Physiology
Background:
- Brachial plexus anesthesia is a common regional anesthetic technique.
- The physiological effects of local anesthetics with epinephrine on cardiovascular parameters require further elucidation.
Purpose of the Study:
- To investigate the impact of brachial plexus anesthesia using lignocaine with adrenaline on cardiac output and peripheral blood flow.
- To assess changes in finger and arm blood flow following the administration of local anesthetic.
Main Methods:
- Utilized electrical impedance plethysmography to measure changes in finger blood flow, arm blood flow, and cardiac output.
- Studied 20 patients undergoing brachial plexus anesthesia with 1% lignocaine and 1:200,000 adrenaline.
Main Results:
- Observed significant increases in cardiac output immediately after effective anesthesia.
- Noted a significant increase in blood flow to the unanesthetized arm.
- Documented a highly significant increase in finger blood flow in the anesthetized hand throughout the procedure.
- Found no overall increase in blood flow to the anesthetized arm.
Conclusions:
- Adrenaline in the local anesthetic solution may increase cardiac output.
- The observed vasoconstriction at the injection site is likely due to adrenaline.
- Peripheral blood flow distribution is altered by brachial plexus anesthesia with adrenaline.