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Open Angle Glaucoma: Treatment01:27

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In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
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Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
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Updated: Nov 7, 2025

Cutaneous Leishmaniasis in the Dorsal Skin of Hamsters: a Useful Model for the Screening of Antileishmanial Drugs
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Ocular Leishmaniasis - A systematic review.

Guillaume Mignot1, Yagnaseni Bhattacharya1, Aravind Reddy1

  • 1Aberdeen Royal Infirmary, NHS Grampian, Scotland.

Indian Journal of Ophthalmology
|April 29, 2021
PubMed
Summary
This summary is machine-generated.

Ocular leishmaniasis (OL) can affect various eye parts, from eyelids to the retina. Prompt diagnosis and aggressive treatment are crucial to prevent severe vision loss from this rare but serious condition.

Keywords:
Amphotericin-BLeishmaniasiscorneaocularuveitis

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Area of Science:

  • Ophthalmology
  • Infectious Diseases
  • Parasitology

Background:

  • Leishmaniasis affects up to 1 million people annually.
  • Ocular leishmaniasis (OL) presentations and treatments are not comprehensively reviewed.
  • A knowledge gap exists regarding the diverse clinical spectrum and management of OL.

Purpose of the Study:

  • To systematically review clinical presentations of ocular leishmaniasis.
  • To summarize the natural course of ocular leishmaniasis.
  • To provide an overview of treatment options for ocular leishmaniasis.

Main Methods:

  • Systematic review of 57 published articles on ocular leishmaniasis cases.
  • Analysis of OL involvement in adnexa, orbit, retina, uvea, and cornea.
  • Synthesis of clinical findings and treatment outcomes.

Main Results:

  • Ocular leishmaniasis involves adnexa, orbit, retina, uvea, and cornea.
  • Palpebral leishmaniasis is often misdiagnosed, potentially leading to chronicity.
  • Retinal involvement presents as self-resolving hemorrhages; uveitis has two main etiologies.
  • Corneal leishmaniasis can be aggressive, leading to perforation, or indolent in the Americas.
  • Systemic antileishmanials are key for keratitis; steroids are vital for uveitis.

Conclusions:

  • Ocular leishmaniasis, though rare, poses a significant threat to vision if untreated.
  • Early diagnosis and aggressive management are essential for favorable outcomes in ocular leishmaniasis.
  • Comprehensive understanding and prompt intervention are critical for managing ocular leishmaniasis effectively.