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Schedule-controlled behavior in infant and juvenile monkeys exposed to lead from birth

D C Rice1

  • 1Toxicology Research Division, Health and Welfare Canada, Ottawa, Ontario.

Neurotoxicology
|January 1, 1988
PubMed

Insights

Lead exposure in infant monkeys (Macaca fascicularis) altered behavior, increasing pauses in fixed ratio tasks and variability in fixed interval tasks, despite no overt toxicity signs.

Area of Science:

  • Neuroscience
  • Toxicology
  • Primate Behavior

Background:

  • Lead acetate exposure in early development can impact neurobehavioral outcomes.
  • Non-human primates offer a valuable model for studying developmental neurotoxicity due to physiological similarities with humans.

Purpose of the Study:

  • To investigate the long-term neurobehavioral effects of early-life lead exposure in cynomolgus monkeys (Macaca fascicularis).
  • To assess the impact of lead acetate on operant conditioning performance across different developmental stages.

Main Methods:

  • Infant cynomolgus monkeys were orally administered lead acetate (2000 micrograms/kg/day) from birth.
  • Behavioral testing included fixed ratio (FR) and fixed interval (FI) schedules of reinforcement starting at 60 days of age.
  • Performance was evaluated at infant and juvenile stages, including a multiple fixed interval-fixed ratio schedule at three years of age.

Main Results:

  • Lead-treated monkeys exhibited increased post-reinforcement pause on FR schedules and decreased pause time on FI schedules during infancy.
  • Juvenile lead-exposed monkeys showed increased FI run rate, pause time, and index of curvature.
  • Elevated performance variability within and between sessions was observed in treated monkeys across multiple measures.

Conclusions:

  • Early-life lead exposure in monkeys induces significant and lasting alterations in operant behavior.
  • These behavioral changes suggest subtle neurodevelopmental disruptions even in the absence of overt clinical signs of toxicity.
  • The study highlights the utility of primate models for understanding the complex behavioral consequences of developmental lead exposure.

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