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Endogenous sex hormones and cardiovascular disease in men. A prospective population-based study

E Barrett-Connor1, K T Khaw

  • 1University of California, San Diego, Department of Community and Family Medicine, La Jolla 92093.

Circulation
|September 1, 1988
PubMed

Insights

This study found no significant link between sex hormones like testosterone and estrogen and cardiovascular disease in older men. Testosterone levels were inversely associated with risk factors, while estrogen showed some positive correlations with metabolic markers.

Area of Science:

  • Endocrinology
  • Cardiovascular Medicine
  • Epidemiology

Background:

  • Endogenous sex hormones are implicated in cardiovascular health.
  • Previous research suggests potential roles for both androgens and estrogens in heart disease risk.
  • Understanding these relationships is crucial for disease prevention and management.

Purpose of the Study:

  • To investigate the association between endogenous sex hormone levels and cardiovascular disease (CVD) in older men.
  • To determine if specific hormones (testosterone, androstenedione, estrone, estradiol) or their ratios predict CVD incidence, mortality, or morbidity.
  • To explore the relationship between sex hormones and established CVD risk factors.

Main Methods:

  • Analysis of stored plasma samples from 1,009 men aged 40-79, collected between 1972-1974.
  • Measurement of endogenous sex hormones and sex hormone-binding globulin (SHBG).
  • 12-year follow-up to assess prevalent and subsequent cardiovascular disease, including mortality and ischemic heart disease.

Main Results:

  • No significant association was found between measured sex hormones (testosterone, androstenedione, estrone, estradiol) and prevalent or subsequent cardiovascular disease, mortality, or morbidity.
  • Sex hormone-binding globulin levels did not differ significantly by disease status.
  • Testosterone showed inverse associations with blood pressure, fasting plasma glucose, triglycerides, and body mass index.
  • Estrone positively correlated with total plasma cholesterol, triglycerides, and glucose.

Conclusions:

  • The study does not support a causal role for elevated endogenous estrogen levels in heart disease.
  • While testosterone levels were inversely related to several metabolic risk factors, this did not translate to a protective association against CVD in this cohort.
  • Further research may be needed to elucidate the complex interplay between sex hormones and cardiovascular health in aging men.

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