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Interference between influenza A viruses with a cleavable and a noncleavable hemagglutinin; pH-stability after mixed

C Scholtissek1, K Müller

  • 1Institut für Virologie, Justus-Liebig-University Giessen, Federal Republic of Germany.

Archives of Virology
|January 1, 1988
PubMed

Insights

Influenza A virus infectivity depends on hemagglutinin (HA) cleavage. Coinfection showed that FPV with cleaved HA acquired noncleaved HA from other viruses, enabling survival at low pH.

Area of Science:

  • Virology
  • Molecular Biology
  • Immunology

Background:

  • Influenza A virus infectivity is determined by hemagglutinin (HA) cleavage.
  • Cleaved HA is sensitive to low pH, while noncleaved HA confers resistance.
  • Fowl plague virus (FPV) possesses cleaved HA, unlike PR 8 or virus N strains.

Purpose of the Study:

  • To investigate the effect of coinfection on influenza A virus HA cleavage and infectivity.
  • To understand the mechanism of HA exchange between influenza viruses.
  • To explore the implications for influenza virus evolution in waterfowl.

Main Methods:

  • Double infection of chick embryo cells with FPV and PR 8 or virus N.
  • Assessment of virus yield and infectivity after low pH treatment.
  • Analysis of HA acquisition through plaque assays in the presence of trypsin.

Main Results:

  • Coinfection significantly reduced the yield of FPV with cleaved HA.
  • Surviving FPV particles acquired resistance to low pH treatment.
  • Evidence of HA exchange, with FPV particles coated by coinfecting virus HA.

Conclusions:

  • Influenza A viruses can exchange hemagglutinin genes during coinfection.
  • This exchange mechanism allows viruses with cleaved HA to acquire resistance to low pH.
  • Findings suggest a role for HA exchange in maintaining nonpathogenic influenza viruses in waterfowl reservoirs.

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