Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Heart Valves01:16

Heart Valves

8.7K
The human heart is a complex organ with an intricate system of valves that regulate blood flow. There are two main types of valves: atrioventricular (AV) valves and semilunar valves.
The AV valves prevent the backflow of blood from the ventricles to the atria during ventricular contraction. These valves function with the assistance of the chordae tendineae and papillary muscles. When the ventricles are relaxed, the chordae tendineae are slack, allowing blood to flow from the atria into the...
8.7K
Mitral Valve Prolapse I: Introduction01:27

Mitral Valve Prolapse I: Introduction

105
IntroductionThe mitral valve, one of the heart's four valves, regulates blood flow. These valves have flaps that open and close to direct blood properly through the heart and body. During each heartbeat, the flaps open for blood to pass through and seal shut to prevent backflow. Specifically, the mitral valve opens to allow blood flow from the heart's upper left chamber to the lower left chamber. It then closes securely as the lower left chamber contracts to pump blood to the body, preventing...
105
Layers of the Heart Wall01:15

Layers of the Heart Wall

4.0K
The heart wall comprises three distinct layers: the epicardium, myocardium, and endocardium. The outermost layer, the epicardium, is the visceral layer of the serous pericardium, featuring a thin, transparent mesothelial surface and an inner layer of areolar connective tissue with fat deposits that increase with age.
The myocardium, the thickest layer, consists of cardiac muscle cells interconnected by intercalated discs and crisscrossing connective tissue fibers. These muscle fibers contract...
4.0K
Chambers of the Heart01:16

Chambers of the Heart

8.1K
The human heart is a complex organ made up of four chambers: the right and left atria and the right and left ventricles. These internal chambers are separated by partitions known as the interatrial and interventricular septa. The exterior of the heart features a groove known as the coronary sulcus that demarcates the atria from the ventricles, while the anterior and posterior interventricular sulci distinguish between the two ventricles.
Deoxygenated blood from the body is received in the right...
8.1K
Mitral Regurgitation I: Introduction01:20

Mitral Regurgitation I: Introduction

112
Mitral regurgitation is characterized by the backward circulation of blood from the left ventricle to the left atrium during systole, a phase of the cardiac cycle when the heart contracts and pumps blood out of the chambers. This abnormal flow occurs primarily due to the dysfunction of the mitral valve or its supporting structures, which include the mitral leaflets, chordae tendineae, annulus, and papillary muscles.Etiology and Mechanisms:Primary Mitral Regurgitation: This type arises from...
112
Location and Orientation of the Heart01:13

Location and Orientation of the Heart

6.2K
The human heart, despite its modest size and weight, is an organ of remarkable strength and endurance. Roughly the size of a fist, the heart weighs between 250 and 350 grams and is nestled within the mediastinum, the medial cavity of the thorax. It extends obliquely for about 12 to 14 cm, resting on the superior surface of the diaphragm. The heart is positioned anterior to the vertebral column and posterior to the sternum, with two-thirds of its mass lying to the left of the midsternal line.
6.2K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Predicting clinically relevant bleeding in new-onset atrial fibrillation patients initiating oral anticoagulant therapy: External validation of the AF-BLEED score.

Thrombosis research·2025
Same author

Sodium-glucose cotransporter 2 inhibitors: a practical guide for the Dutch cardiologist based on real-world experience.

Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation·2021
Same author

Recurrent hypocalcaemic torsades de pointes due to hungry bone syndrome: a rare complication of thyroidectomy.

Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation·2021
Same author

Too much or not enough; balancing insulin levels in a diabetic patient sent for <sup>18</sup>F-FDG PET/CT of prosthetic heart valve endocarditis.

Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology·2019
Same author

Reproducibility of right-to-left shunt quantification using transthoracic contrast echocardiography in hereditary haemorrhagic telangiectasia.

Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation·2018
Same author

SMAD4 gene mutation increases the risk of aortic dilation in patients with hereditary haemorrhagic telangiectasia.

International journal of cardiology·2017

Related Experiment Video

Updated: Oct 29, 2025

A Pulmonary Trunk Banding Model of Pressure Overload Induced Right Ventricular Hypertrophy and Failure
06:47

A Pulmonary Trunk Banding Model of Pressure Overload Induced Right Ventricular Hypertrophy and Failure

Published on: November 29, 2018

9.9K

Left ventricular false tendons.

S Velthuis1, P J Senden2

  • 1Department of Cardiology, Meander Medical Centre, Amersfoort, The Netherlands. s.velthuis@meandermc.nl.

Netherlands Heart Journal : Monthly Journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation
|July 7, 2021
PubMed
Summary

Left ventricular false tendons (LVFTs) are common, normal heart variants. Despite some concerns, individuals with LVFTs do not face an increased risk of sudden cardiac death or arrhythmias.

Keywords:
Abnormal cardiac remodellingLeft ventricular false tendonVentricular arrhythmias

More Related Videos

Transthoracic Speckle Tracking Echocardiography for the Quantitative Assessment of Left Ventricular Myocardial Deformation
09:05

Transthoracic Speckle Tracking Echocardiography for the Quantitative Assessment of Left Ventricular Myocardial Deformation

Published on: October 20, 2016

19.8K
Rat Model of Right-Sided Cardiac Remodeling and Arrhythmia Using Pulmonary Artery Banding
10:39

Rat Model of Right-Sided Cardiac Remodeling and Arrhythmia Using Pulmonary Artery Banding

Published on: August 30, 2024

950

Related Experiment Videos

Last Updated: Oct 29, 2025

A Pulmonary Trunk Banding Model of Pressure Overload Induced Right Ventricular Hypertrophy and Failure
06:47

A Pulmonary Trunk Banding Model of Pressure Overload Induced Right Ventricular Hypertrophy and Failure

Published on: November 29, 2018

9.9K
Transthoracic Speckle Tracking Echocardiography for the Quantitative Assessment of Left Ventricular Myocardial Deformation
09:05

Transthoracic Speckle Tracking Echocardiography for the Quantitative Assessment of Left Ventricular Myocardial Deformation

Published on: October 20, 2016

19.8K
Rat Model of Right-Sided Cardiac Remodeling and Arrhythmia Using Pulmonary Artery Banding
10:39

Rat Model of Right-Sided Cardiac Remodeling and Arrhythmia Using Pulmonary Artery Banding

Published on: August 30, 2024

950

Area of Science:

  • Cardiology
  • Cardiac Anatomy
  • Electrophysiology

Background:

  • Left ventricular false tendons (LVFTs) are fibromuscular structures within the heart.
  • Their association with exercise safety, arrhythmias, and cardiac remodeling is debated.
  • Improved imaging has revealed LVFTs are more prevalent than previously thought.

Purpose of the Study:

  • To clarify the clinical significance of left ventricular false tendons.
  • To evaluate the association between LVFTs and adverse cardiac events.
  • To determine if LVFTs represent a normal variant or a pathological finding.

Main Methods:

  • Review of cardiac imaging studies.
  • Analysis of electrocardiographic findings.
  • Correlation with clinical outcomes and exercise history.

Main Results:

  • LVFTs are frequently observed, suggesting they are a common anatomic variant.
  • Electrocardiographic abnormalities can be associated with LVFTs.
  • No increased risk of ventricular arrhythmias or sudden cardiac death was found in individuals with LVFTs.

Conclusions:

  • Left ventricular false tendons should be considered a normal anatomical variant.
  • The presence of LVFTs does not appear to confer an increased risk for serious cardiac events.
  • Further research may clarify the role of LVFTs in specific clinical contexts.