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Related Concept Videos

Aneurysm III: Interprofessional Care01:26

Aneurysm III: Interprofessional Care

64
Aneurysm management involves either conservative medical therapy or surgical intervention, depending on the size and symptoms of the aneurysm. Conservative management is generally reserved for smaller, asymptomatic aneurysms, while larger or symptomatic aneurysms often necessitate surgical repair.Conservative Medical TherapyFor small, asymptomatic aneurysms, particularly abdominal aortic aneurysms (AAA) less than 5.5 centimeters in diameter, conservative medical therapy is recommended. This...
64
Aneurysm IV: Nursing Management01:22

Aneurysm IV: Nursing Management

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Vigilant monitoring for aneurysm rupture is essential for patients undergoing aortic surgery.Preoperative Nursing ManagementContinuously monitor the patient for manifestations of aneurysm rupture, such as pallor, weakness, tachycardia, hypotension, abdominal, back, groin, or periumbilical pain, changes in consciousness, and a pulsating abdominal mass. Regularly assess the patient's peripheral pulses.Instruct the patient to consume a clear liquid diet the day before surgery and administer...
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Aneurysm I: Introduction01:30

Aneurysm I: Introduction

88
An aortic aneurysm is a localized outpouching or dilation at a weak point in the artery wall. It may involve different parts of the aorta, such as the abdominal aorta, aortic arch, or thoracic aorta.Etiological factorsSeveral disorders are associated with aortic aneurysms.Congenital causes, such as primary connective tissue disorders like Marfan syndrome, impact the integrity and strength of connective tissues, notably affecting the aorta. Marfan syndrome is a genetic disorder that specifically...
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Aneurysm II: Clinical Manifestations and Diagnostic Studies01:21

Aneurysm II: Clinical Manifestations and Diagnostic Studies

58
Thoracic, aortic arch and abdominal aneurysms are significant vascular conditions that can present with various clinical manifestations and lead to serious complications. Understanding these manifestations and the appropriate diagnostic studies is essential for effective management and treatment.Thoracic Aortic AneurysmsThoracic aortic aneurysms often remain asymptomatic until they reach a size that impinges on adjacent structures. They typically cause deep, diffuse chest pain that radiates to...
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Related Experiment Video

Updated: Oct 29, 2025

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage
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Pituitary dysfunction after aneurysmal subarachnoidal hemorrhage.

Susanna Bacigaluppi1, Chiara Robba2, Nicola L Bragazzi3

  • 1Department of Neurosurgery, E.O. Ospedali Galliera, Genova, Italy.

Handbook of Clinical Neurology
|July 9, 2021
PubMed
Summary
This summary is machine-generated.

Neuroendocrine dysfunction is common after aneurysmal subarachnoid hemorrhage (aSAH), with rates varying widely in acute and chronic phases. More research is needed to standardize diagnosis and treatment for these debilitating conditions.

Keywords:
Aneurysmal subarachnoid hemorrhageNeuroendocrinologyNeuropsychologic sequelaePituitary dysfunction

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Last Updated: Oct 29, 2025

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Area of Science:

  • Neuroendocrinology
  • Neurosurgery
  • Critical Care Medicine

Background:

  • Aneurysmal subarachnoid hemorrhage (aSAH) is a severe neurological condition with significant morbidity.
  • Neuroendocrine impairment is frequently observed following aSAH, but prevalence estimates vary considerably.

Purpose of the Study:

  • To review the prevalence and patterns of neuroendocrine dysfunction in patients with aSAH.
  • To highlight the need for standardized diagnostic and therapeutic approaches.

Main Methods:

  • Systematic review of existing literature on neuroendocrine dysfunction in aSAH.
  • Analysis of prevalence rates based on time of assessment (acute vs. chronic) and diagnostic criteria.

Main Results:

  • Acute aSAH shows highly variable neuroendocrine impairment (3.8%–92.3%).
  • Chronic aSAH reveals overall impairment rates of 47%–83.3%, with specific deficiencies ranging from 2.5%–83.3%.
  • Pituitary deficiency tends to decrease over time, with some recovery and new dysfunctions emerging.

Conclusions:

  • Neuroendocrine dysfunction is prevalent in aSAH patients, impacting recovery and quality of life.
  • Current findings are based on heterogeneous data, necessitating high-quality studies with standardized protocols.
  • Routine endocrine screening and hormone replacement therapy require further evidence before implementation in clinical guidelines.