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Hemichorea due to ipsilateral thalamic infarction: A case report
Zhao-Sheng Li1, Jia-Jia Fang1, Xiao-Hui Xiang1
1Department of Neurology, The Fourth Affiliated Hospital, Zhejiang University School of Medicine, Yiwu 322000, Zhejiang Province, China.
Ipsilateral hemichorea, involuntary movements on the same side as the brain lesion, can occur after a thalamic stroke. This rare condition may be explained by the thalamus's role in bilateral motor control.
Area of Science:
- Neurology
- Neuroscience
Background:
- Hemichorea typically arises from basal ganglia vascular lesions, usually contralateral to affected limbs.
- The pathophysiology of ipsilateral hemichorea remains poorly understood.
- This review explores mechanisms of ipsilateral hemichorea following cerebral infarction.
Observation:
- A 72-year-old woman presented with sudden onset left-sided hemichorea and mild right-sided hemiparesis.
- Involuntary movements spread to involve the left limbs.
- MRI revealed a left thalamic infarction.
Findings:
- The patient's hemichorea resolved with haloperidol treatment.
- Hemiparesis improved with rehabilitation physiotherapy.
- Ipsilateral hemichorea following thalamic infarction is a rare but documented phenomenon.
Implications:
- The thalamus, a key relay station, exerts bilateral control over motor function.
- Understanding these pathways may explain ipsilateral hemichorea.
- This case highlights the complex relationship between thalamic lesions and motor control deficits.
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