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Response to intraperitoneal Staphylococcus epidermidis challenge in renal failure mice

B Gallimore1, R F Gagnon, G K Richards

  • 1Department of Medicine, Montreal General Hospital, McGill University, Quebec, Canada.

Kidney International
|November 1, 1987
PubMed

Insights

Renal failure significantly delays Staphylococcus epidermidis clearance and diminishes the inflammatory response in peritonitis, impacting end-stage renal disease patients undergoing continuous ambulatory peritoneal dialysis.

Area of Science:

  • Nephrology
  • Infectious Diseases
  • Immunology

Background:

  • End-stage renal disease (ESRD) patients on continuous ambulatory peritoneal dialysis (CAPD) are susceptible to Staphylococcus epidermidis peritonitis.
  • Renal failure's impact on the pathogenesis of S. epidermidis peritonitis requires further elucidation.

Purpose of the Study:

  • To investigate the role of renal failure in the pathogenesis of S. epidermidis peritonitis in a mouse model.
  • To assess bacterial clearance and inflammatory responses in mice with surgically-induced renal failure.

Main Methods:

  • Surgically-induced renal failure in mice, followed by intraperitoneal (i.p.) inoculation of S. epidermidis.
  • Assessment of bacterial clearance and peritoneal inflammatory response over 72 hours.
  • Comparison between renal failure mice, sham-operated controls, and normal controls.

Main Results:

  • Peritoneal clearance of S. epidermidis was significantly delayed in renal failure mice.
  • Viable bacteria persisted in the peritoneum even after negative peritoneal washings.
  • Peritoneal inflammatory response, particularly polymorphonuclear cell influx, was markedly diminished in renal failure mice.

Conclusions:

  • Renal failure impairs the local peritoneal inflammatory response and delays bacterial clearance in S. epidermidis peritonitis.
  • The precise factors contributing to this impairment in renal failure mice need further investigation.

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