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Published on: August 18, 2016
Multivessel coronary artery vasospasm masquerading as surgical coronary atherosclerotic disease
1Department of Cardiothoracic Surgery, Auckland City Hospital, Auckland, New Zealand.
Insights
A case of severe coronary vasospasm mimicking obstructive coronary artery disease was successfully treated with thoracic sympathectomy. This intervention managed refractory vasospastic angina, offering a novel therapeutic approach.
Area of Science:
- Cardiology
- Vascular Medicine
- Surgical Innovation
Background:
- A 39-year-old female presented with escalating angina symptoms.
- Initial assessments suggested obstructive coronary artery disease requiring surgical intervention.
Observation:
- Coronary angiography revealed significant vasospasm in multiple coronary arteries and bypass grafts, not obstructive disease.
- The patient experienced cardiac arrest during surgery, necessitating extracorporeal membrane oxygenation (ECMO).
Findings:
- Severe multivessel coronary vasospasm was diagnosed as the underlying cause of the patient's symptoms.
- Medical management with calcium channel blockers and bilateral thoracic sympathectomy effectively controlled the vasospasm.
Implications:
- This case underscores the critical importance of considering coronary vasospasm in angina diagnosis, even with apparent obstructive findings.
- Thoracic sympathectomy presents a potentially novel and effective treatment for refractory vasospastic angina.
Background And Aim:
We report a case of a 39-year-old lady presenting with worsening angina.
Materials And Methods:
This is a case report study. Clinical case data was retrieved from hospital paper and electronic records.
Results:
Invasive coronary angiography revealed disease in the left main stem, proximal left anterior descending (LAD) artery and circumflex artery. The patient proceeded to on-pump coronary artery bypass grafting. Intraoperatively, there were multiple unsuccessful attempts to wean off cardiopulmonary bypass. An on-table angiogram-which initially triggered asystole requiring internal cardiac massage and institution of venoarterial extracorporeal membrane oxygenation (ECMO)- showed no native coronary artery disease. Instead, this angiogram revealed severe vasospasm with narrowing in the grafts and distal LAD. The patient received calcium channel blockers and bilateral thoracic sympathectomies to suppress any further coronary vasospasm. She was subsequently successfully weaned off ECMO.
Discussion And Conclusion:
This case reveals the life-threatening nature and diagnostic dilemma posed by severe multivessel coronary vasospasm. We also highlight the novel role of thoracic sympathectomy for definitive management of refractory vasospastic angina.
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