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Leptospiral attachment to extracellular matrix of mouse fibroblast (L929) cells

T Ito1, R Yanagawa

  • 1Department of Hygiene and Microbiology, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Japan.

Veterinary Microbiology
|October 1, 1987
PubMed

Insights

Highly virulent Leptospira strains attach better to the extracellular matrix (ECM) than less virulent ones. This attachment can be inhibited by specific immunoglobulin G Fab fragments, suggesting a role in leptospiral infection.

Area of Science:

  • Microbiology
  • Pathogen-Host Interactions
  • Extracellular Matrix Biology

Background:

  • Leptospirosis is a zoonotic disease caused by Leptospira bacteria.
  • The extracellular matrix (ECM) plays a role in tissue structure and pathogen adhesion.
  • Understanding pathogen attachment mechanisms is crucial for disease control.

Purpose of the Study:

  • To investigate the attachment of Leptospira interrogans to the host's extracellular matrix (ECM).
  • To determine if bacterial virulence correlates with ECM attachment.
  • To explore potential inhibition mechanisms for Leptospira attachment to ECM.

Main Methods:

  • Mouse fibroblast (L929) cells were cultured on coverslips and solubilized to create an ECM.
  • Highly virulent, intermediately virulent, and avirulent strains of Leptospira interrogans (serovars copenhageni, canicola, pomona) were incubated with the ECM.
  • The attachment efficiency of different Leptospira strains to the ECM was quantified.
  • The effect of homologous immunoglobulin G Fab fragments on the attachment of highly virulent L. interrogans copenhageni to ECM was assessed.

Main Results:

  • Highly virulent strains of Leptospira interrogans (serovars copenhageni, canicola, and pomona) demonstrated significantly greater attachment to the ECM compared to intermediately virulent and avirulent strains.
  • A positive correlation was observed between the virulence of Leptospira strains and their ability to attach to the ECM.
  • The presence of homologous immunoglobulin G Fab fragments effectively inhibited the attachment of highly virulent L. interrogans copenhageni to the ECM.

Conclusions:

  • Bacterial virulence in Leptospira interrogans is associated with enhanced attachment to the host's extracellular matrix.
  • The ECM serves as a significant attachment substrate for virulent Leptospira strains.
  • Immunoglobulin G Fab fragments targeting specific bacterial components show potential for inhibiting Leptospira ECM attachment, suggesting therapeutic implications.

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