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Amplifiers in Tinnitus Patients
Henk M Koning1, Bas C Ter Meulen2
1Department of Pain therapy, Pain Clinic De Bilt, De Bilt, The Netherlands.
Introduction:
High tinnitus loudness can be provoked by peripheral disorders of the somatosensory and/or auditory system.
Objectives:
The object of our study was to compare high tinnitus loudness patients with low tinnitus loudness patients and to find specific factors associated with high tinnitus loudness.
Design:
A retrospective cohort analysis of 234 patients with tinnitus as main complaint who visited our clinic in a four-year period.
Methods:
Data obtained from the subjects were age, sex, the loudness of tinnitus estimated by the VAS, and the outcomes of the audiogram and the cervical spine radiograph.
Results:
High tinnitus loudness was associated with a higher prevalence of females, more dizziness, less self-perceived hearing loss, more cervical disc degeneration at C3 to C6, and a larger anterior spur of cervical vertebrae C3 to C6. Females had a lower prevalence of tinnitus but a higher chance on high tinnitus loudness. Gender, the size of the largest anterior spur from C3 to C6, and the ratio of hearing loss at 8 kHz and hearing loss at 2 kHz are involved in the amplification of tinnitus loudness.
Conclusions:
High tinnitus loudness can be provoked by peripheral disorders of the somatosensory and/or auditory system. A steep audiometric edge between hearing at 2 kHz and hearing at 8 kHz and/or cervical spine pathology with sympathetic nervous system irritation can amplify tinnitus loudness causing high tinnitus loudness.
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