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[Pulmonary calcinosis].

P Carles1, G Dongay, D Lauque

  • 1Service de Médecine, CHU Toulouse-Purpan.

Revue Des Maladies Respiratoires
|January 1, 1987
PubMed
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Hydrochlorothiazide treatment can unmask underlying hyperparathyroidism, leading to interstitial pulmonary calcinosis. This case highlights the importance of monitoring calcium levels during diuretic therapy.

Area of Science:

  • Nephrology
  • Pulmonology
  • Endocrinology

Background:

  • Hydrochlorothiazide is a commonly prescribed diuretic used to treat hypertension and edema.
  • Hypercalcemia, elevated calcium levels in the blood, can have various underlying causes.
  • Secondary hyperparathyroidism involves overactivity of the parathyroid glands due to other conditions.

Observation:

  • A 56-year-old male presented with hypercalcemia after several months of hydrochlorothiazide therapy.
  • Interstitial pulmonary calcinosis, calcium deposits in the lungs, was diagnosed.
  • The patient had a history of quiescent parathyroid adenomatous cysts.

Findings:

  • Hydrochlorothiazide therapy likely triggered the manifestation of previously asymptomatic secondary hyperparathyroidism.

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  • The hyperparathyroidism, caused by parathyroid adenomatous cysts, led to hypercalcemia.
  • Hypercalcemia resulted in interstitial pulmonary calcinosis.

    • Implications:

    • This case underscores the potential for hydrochlorothiazide to unmask latent endocrine disorders.
    • Close monitoring of calcium levels is crucial in patients receiving long-term hydrochlorothiazide treatment.
    • Understanding drug-induced hypercalcemia is vital for accurate diagnosis and management of pulmonary complications.