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Tumour necrosis factor-like activity on paraformaldehyde-fixed monocyte monolayers

T Espevik1, J Nissen-Meyer

  • 1Institute of Cancer Research, University of Trondheim, Norway.

Immunology
|August 1, 1987
PubMed

Insights

Tumor necrosis factor (TNF)-related proteins are found on monocyte membranes, causing cell death. Gamma-interferon activation increases this TNF-like activity on monocytes.

Area of Science:

  • Immunology
  • Cell Biology

Background:

  • Tumor necrosis factor (TNF) is a key inflammatory cytokine.
  • Monocytes play a crucial role in the immune response.

Purpose of the Study:

  • To investigate the presence and activity of TNF-related proteins on the monocyte surface.
  • To determine the effect of gamma-interferon (gamma-IFN) activation on monocyte-associated TNF-like activity.

Main Methods:

  • Paraformaldehyde-fixed monocyte monolayers were used to assess cytotoxic activity against WEHI 164 clone 13 cells.
  • Neutralizing antisera against recombinant TNF (rTNF) and cytotoxic factor (CF) were employed for inhibition studies.
  • Crude monocyte membrane preparations were analyzed for cytotoxic activity and characterized by filtration.
  • Comparison of TNF-like activity between activated and unactivated monocytes.

Main Results:

  • Fixed monocytes induced cytolysis of WEHI 164 clone 13 cells, indicating TNF-related activity.
  • Cytolysis was inhibited by antisera against rTNF and CF, confirming the involvement of TNF.
  • Cytotoxic activity was associated with particulate structures in monocyte membrane preparations.
  • Gamma-IFN-activated monocytes exhibited significantly higher TNF-like activity compared to unactivated monocytes.

Conclusions:

  • A TNF-related protein is associated with the monocyte surface membrane.
  • Monocyte-derived cytotoxic factor (CF) is likely identical to natural TNF.
  • Gamma-IFN activation enhances the TNF-like cytotoxic activity of monocytes.

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