Corrigendum to "MicroRNA-30-5p Suppresses Inflammatory Factor-Induced Endothelial Cell Injury by Targeting TCF21"

Zhenyu Zhou1,2, Yu Chen3, Dongying Zhang1

  • 1Department of Cardiology, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, China.

Mediators of Inflammation
|November 1, 2021
PubMed

Insights

This study corrects a previous article DOI. The corrected DOI is 10.1155/2019/1342190 for enhanced discoverability and citation accuracy.

Area of Science:

  • Scientific publishing
  • Digital object identifiers
  • Bibliometrics

Context:

  • Correction of a previously published article DOI.
  • Ensuring accurate citation and retrieval of scientific literature.
  • Maintaining the integrity of the scientific record.

Purpose:

  • To provide the correct Digital Object Identifier (DOI) for a specific scientific article.
  • To facilitate accurate referencing and access to the research.
  • To rectify a citation error and improve scholarly communication.

Summary:

  • The article DOI has been corrected to 10.1155/2019/1342190.
  • This correction ensures that readers and indexing services can accurately locate the intended publication.
  • The update supports the proper attribution of scientific work.

Impact:

  • Improved accessibility of the scientific article.
  • Enhanced accuracy in academic citations and bibliographies.
  • Reduced confusion and potential errors in literature searches.

Related Concept Videos

Regulation of Angiogenesis and Blood Supply01:24

Regulation of Angiogenesis and Blood Supply

Rapidly dividing tumors, embryos, and wounded tissues require more oxygen than usual, lowering the oxygen concentration in the blood. At low oxygen or hypoxic conditions, an oxygen-sensitive transcription factor called the hypoxia-inducible factor 1 or HIF1 is activated. HIF1 is a dimeric protein of alpha (ɑ) and beta (β) subunits.  Under optimal oxygen conditions, HIF1β is present in the nucleus while HIF1ɑ remains in the cytosol. HIF1ɑ is hydroxylated by prolyl...
2.9K
TGF - β Signaling Pathway01:16

TGF - β Signaling Pathway

The TGF-β signaling pathway regulates cell growth, differentiation, adhesion, motility, and development. TGF-β ligands that induce TGF-β signaling are synthesized in their latent form. Several proteases or cell surface receptors such as integrins act upon the latent form, releasing the active ligand. There are three types of mammalian TGF-βs: (TGF-β1, TGF-β2, and TGF-β3) that bind as homodimers or heterodimers to TGF-β receptors. The TGF-β receptors...
8.0K
NF-κB-dependent Signaling Pathway02:26

NF-κB-dependent Signaling Pathway

The transcription factor NF-κB was discovered in 1986 in the lab of Nobel laureate Professor David Baltimore, for its interaction with the immunoglobulin light chain enhancer in B-cells. After more than three decades of study, it is now evident that NF-κB regulates the expression of over 100 genes. Most of these genes play an essential role in the innate and adaptive immune responses as well as the inflammatory responses of animals.
NF-κB-dependent Signaling Mechanism
The...
7.9K
Inflammatory Response I: Vascular and Cellular01:30

Inflammatory Response I: Vascular and Cellular

The inflammatory response is the body's defense against infection, injury, or irritation from bacteria, trauma, toxins, or heat. Inflammation helps locate and destroy pathogens and remove damaged tissue elements to heal the body. During this initial phase, fluid, blood products, and nutrients migrate to the injured area, resulting in redness, heat, swelling, ache, and loss of function. Moreover, signs of systemic inflammation include fever, increased WBC count, malaise, anorexia, nausea,...
13.1K